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61,005 resultsShowing papers similar to Impact of sub-chronic polystyrene nanoplastics exposure on hematology, histology, and endoplasmic reticulum stress-related protein expression in Nile tilapia (Oreochromis niloticus)
ClearSub-chronic exposure of Oreochromis niloticus to environmentally relevant concentrations of smaller microplastics: Accumulation and toxico-physiological responses
Researchers exposed Nile tilapia to low, environmentally relevant concentrations of polystyrene microplastics for 14 days and found the particles accumulated in multiple organs including the brain, liver, and reproductive tissues. The fish showed changes in blood chemistry, increased stress hormones, and signs of liver and kidney dysfunction. These results suggest that even realistic levels of microplastic pollution can cause measurable physiological harm in fish.
Ecotoxicological effects of polystyrene nanoplastics on common carp: Insights into blood parameters, DNA damage, and gene expression
Exposing common carp to polystyrene nanoplastics caused significant DNA damage in blood and brain cells, along with changes in genes related to immune function and stress response. Higher concentrations led to more severe effects, and the nanoplastics also disrupted liver antioxidant defenses. Since carp are widely consumed fish, these findings raise questions about the safety of fish from nanoplastic-contaminated waters for human consumption.
Toxicological effects of nano- and micro-polystyrene plastics on red tilapia: Are larger plastic particles more harmless?
Researchers exposed red tilapia to three sizes of polystyrene particles (0.3, 5, and 70-90 micrometers) to compare their toxic effects. The study found that the largest particles showed the highest accumulation in tissues, but all sizes induced oxidative stress, disrupted cytochrome P450 enzymes, caused neurotoxicity, and altered metabolic profiles, indicating that even smaller nanoplastics can cause significant harm to fish.
Deleterious Effects of Polypropylene Microplastic Ingestion in Nile Tilapia (Oreochromis niloticus)
Researchers fed Nile tilapia daily doses of polypropylene microplastics for 30 days and observed significant health effects including changes in blood cell counts, altered gut bacteria, and tissue damage to the intestines and liver. The higher dose group showed more pronounced effects, including elevated inflammatory markers and signs of oxidative stress. The study provides evidence that chronic ingestion of microplastics commonly found in aquatic environments can cause meaningful harm to a widely consumed fish species.
Nano polystyrene microplastics could accumulate in Nile tilapia (Oreochromis niloticus): Negatively impacts on the intestinal and liver health through water exposure
Researchers exposed Nile tilapia fish to polystyrene microplastics of different sizes (ranging from 80 nanometers to 80 micrometers) and found that the smallest particles were most likely to accumulate in the body. The 80-nanometer particles caused the most severe damage to intestinal and liver tissues, disrupting cell growth and triggering inflammation and oxidative stress. The study suggests that nanoscale plastic particles may pose greater health risks to fish than larger microplastics.
Sub-chronic nanoplastic toxicity in Etroplus suratensis (Pisces, Cichilidae): Insights into tissue accumulation, stress and metabolic disruption
Researchers exposed pearl spot fish to polystyrene nanoplastics at different concentrations for 14 days and found that the particles accumulated in multiple organs with concentration-dependent distribution patterns. The nanoplastics caused elevated glucose and cholesterol levels, suppressed antioxidant defenses, and increased markers of oxidative damage and stress. Gene expression changes in stress response and growth-related genes suggest that nanoplastic exposure may impair both immune function and normal development in fish.
Effects of polystyrene nanoplastics on oxidative stress, histopathology and intestinal microbiota in largemouth bass (Micropterus salmoides)
Researchers exposed largemouth bass — a commercially important freshwater fish — to polystyrene nanoplastics (tiny plastic particles 100 nanometers in size) for up to 19 days, finding tissue damage in the gills, liver, and intestines along with elevated markers of cellular stress. While growth was not significantly affected, the fish adjusted their gut microbiome in response, suggesting nanoplastics trigger adaptive but potentially harmful physiological changes.
Polystyrene nanospheres-induced hepatotoxicity in swamp eel (Monopterus albus): From biochemical, pathological and transcriptomic perspectives
Researchers exposed swamp eels to polystyrene nanoplastics for 28 days and found significant liver damage including oxidative stress, tissue abnormalities, and disrupted gene expression related to immune response and metabolism. Higher concentrations caused more severe liver injury, with changes detectable at both the biochemical and genetic levels. This study adds evidence that nanoplastic exposure can harm liver function in freshwater species important to aquaculture and local food supplies.
Microplastics and nanoplastics induced differential respiratory damages in tilapia fish Oreochromis niloticus
Researchers exposed tilapia fish to polystyrene particles of three sizes at environmentally realistic levels and found that all sizes caused respiratory damage, with the medium and large microplastics causing more severe breathing problems than the smallest nanoparticles. The microplastics disrupted energy production in gill tissue and triggered immune responses. Since tilapia is a widely farmed and consumed fish, these findings raise concerns about both fish welfare in plastic-contaminated waters and the quality of farmed fish as food.
Long-Term Exposure to Polystyrene Nanoplastics Impairs the Liver Health of Medaka
Researchers found that three months of exposure to polystyrene nanoplastics caused significant liver damage in medaka fish, including oxidative stress, immune disruption, and altered gene expression related to lipid metabolism and detoxification pathways.
Accumulation, tissue distribution, and biochemical effects of polystyrene microplastics in the freshwater fish red tilapia (Oreochromis niloticus)
Researchers exposed freshwater tilapia to polystyrene microplastics at varying concentrations for two weeks and tracked where the particles accumulated in the body. The microplastics concentrated primarily in the gut and gills, but also reached the liver and brain, with accumulation increasing over time and at higher doses. The study found that the particles caused oxidative stress and altered enzyme activity in the fish, indicating that even short-term microplastic exposure can trigger measurable biological harm in freshwater species.
The Effect of Exposure to Microplastic Polystyrene (PS) in Feed on the Haematology of Tilapia (Oreochromis niloticus)
Researchers fed tilapia fish diets containing polystyrene microplastics to study the effects on blood health indicators. They found that microplastic exposure altered several blood parameters in the fish, indicating physiological stress. The study matters because tilapia is a widely consumed fish species, and the results suggest that microplastic contamination in aquatic environments could affect both fish health and food safety.
Effect of Polystyrene Microplastics on the Antioxidant System and Immune Response in GIFT (Oreochromis niloticus)
Researchers exposed farmed tilapia to polystyrene microplastics of various sizes and found that smaller particles caused more oxidative stress and stronger immune responses. Fish exposed to nanometer-sized plastics showed higher levels of reactive oxygen species and inflammatory markers compared to those exposed to larger particles. The study suggests that the size of microplastics matters significantly when evaluating their impact on fish health.
Growth performance, hematological and oxidative stress responses in Nile tilapia (Oreochromis niloticus) exposed to polypropylene microplastics
Nile tilapia fish fed polypropylene microplastics for 7 weeks showed significantly reduced growth, blood abnormalities indicating anemia, and oxidative stress damage in both liver and brain tissue. The effects were dose-dependent, with higher microplastic concentrations causing more severe harm. Since tilapia is one of the most widely farmed and consumed fish globally, these findings raise concerns about the health of both farmed fish and the people who eat them.
Polystyrene Nanoplastics Induce Multi-Organ Toxicity in the Rainbow Trout (Oncorhynchus mykiss): An Integrated Assessment of Physiological, Immunological, and Molecular Responses
Rainbow trout were exposed to polystyrene nanoplastics at three concentrations for 28 days and assessed for physiological, immunological, and molecular responses across multiple organs. NPs accumulated in liver, spleen, and intestine, causing dose-dependent oxidative stress, immune dysregulation, and altered gene expression, demonstrating multi-organ toxicity in a commercially important fish species.
Exploration of polyacrylamide microplastics and evaluation of their toxicity on multiple parameters of Oreochromis niloticus
Researchers evaluated the toxicity of polyacrylamide microplastics on Nile tilapia fish at different concentrations and found significant harmful effects across multiple biological systems. The microplastics disrupted antioxidant enzymes, reduced blood cell counts, and caused histological damage to gills, liver, and intestine. The study indicates that polyacrylamide microplastics, increasingly present in aquatic environments, are toxic agents with broad physiological impacts on freshwater fish.
Effects of Sub-Chronic Exposure to Polystyrene Nanoplastics on Lipid and Antioxidant Metabolism in Sparus aurata
Researchers exposed gilthead seabream to polystyrene nanoplastics for 14 days and measured effects on blood, tissue, and gene expression. While the fish showed no visible tissue damage or changes in body condition, they had reduced hemoglobin levels and significant downregulation of genes related to fat metabolism, growth, and antioxidant defense. The study suggests that nanoplastics can cause subtle but meaningful biological changes in fish even when outward signs of harm are absent.
Transcriptome sequencing and metabolite analysis reveal the toxic effects of nanoplastics on tilapia after exposure to polystyrene
Researchers exposed larval tilapia to polystyrene nanoplastics and then analyzed changes in gene expression and metabolic profiles after a recovery period. They found that nanoplastic exposure disrupted immune-related pathways, energy metabolism, and lipid processing in the fish, with some effects persisting even after exposure ended. The study suggests that nanoplastics can cause lasting metabolic and immune disruptions in freshwater fish.
Polystyrene microparticles can affect the health status of freshwater fish – Threat of oral microplastics intake
Researchers fed juvenile rainbow trout polystyrene microplastics at three dietary concentrations for six weeks and assessed multiple health parameters. They found that the highest concentration triggered immune responses, liver and gill damage, disrupted antioxidant balance, and reduced plasma proteins. The study demonstrates that oral microplastic intake can negatively affect the health of freshwater fish across multiple organ systems.
Tissue damage, antioxidant capacity, transcriptional and metabolic regulation of red drum Sciaenops ocellatus in response to nanoplastics exposure and subsequent recovery
Researchers exposed red drum fish to polystyrene nanoplastics for seven days and then monitored their recovery over two weeks. They found persistent liver and gill tissue damage along with ongoing oxidative stress even after the nanoplastics were removed from the water. The study suggests that nanoplastic exposure can cause lasting harm to marine fish that does not quickly reverse once the exposure ends.
Toxicological effects of polystyrene nanoplastics on marine organisms
Researchers exposed Pacific white shrimp to polystyrene nanoplastics at various concentrations and measured immune, antioxidant, and tissue responses after seven days. They found that nanoplastic exposure disrupted immune function, increased oxidative stress, and caused tissue damage, particularly in the hepatopancreas and gills. The study adds to growing evidence that nanoplastics can harm the health of commercially important marine species.
The Effect of Exposure to Polystyrene Nanoplastics on Cytokine Levels and Reproductive System of Male Tilapia
Researchers fed male tilapia fish different doses of polystyrene nanoplastics for 25 days and examined the effects on their reproductive systems. While inflammatory markers in the blood were not significantly affected, the nanoplastics caused a notable reduction in the number of sperm-producing cells in testicular tissue. The study suggests that nanoplastic exposure may pose risks to reproductive health in fish even without triggering obvious immune responses.
Bioaccumulation and homeostatic alterations in trout exposed to a sublethal dose of polystyrene nanoplastics
Researchers orally exposed rainbow trout to polystyrene nanoplastics and found the particles accumulated mainly in the gut and blood — not the liver — causing subtle immune and metabolic changes without visible tissue damage after 96 hours. These findings suggest nanoplastics selectively distribute in fish tissues and trigger mild biological responses even at sublethal doses.
Effects of Polystyrene Nanoplastics on Oxidative Stress, Blood Biochemistry, and Digestive Enzyme Activity in Goldfish (Carassius auratus)
Goldfish exposed to polystyrene nanoplastics in their diet for 21 days showed significant oxidative stress, disrupted blood chemistry, and reduced digestive enzyme activity, with effects worsening at higher doses. The smallest nanoplastics caused the most damage to the fishes' antioxidant defense systems and overall health. These findings add to the evidence that nanoplastics in aquatic environments can harm fish health in ways that may affect the safety of fish consumed by humans.