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61,005 resultsShowing papers similar to Histopathological evaluation of two Blennius fishes exposed to microplastics via feeding
ClearHistological and Histochemical Effects of Microplastics Administration in Oreochromis niloticus Fingerlings
Researchers exposed Nile tilapia to two types of microplastics and examined histological and histochemical changes in gills, liver, and kidneys, finding tissue-level damage that demonstrates the harmful effects of microplastic ingestion on vital fish organs.
Microplastics induced histopathological lesions in some tissues of tilapia (Oreochromis niloticus) early juveniles
Researchers exposed young tilapia fish to different concentrations of microplastics for 15 days to study effects on their organs. The study found damage across multiple tissues including the kidney, liver, pancreas, gills, and muscles, with effects ranging from inflammation and cell death to structural deformation. These findings suggest that microplastic exposure can cause widespread organ damage in fish even at relatively low concentrations.
Toxicological Assessment of Microplastics in Zebrafish: Biochemical Responses and Histopathological Changes
Zebrafish were exposed to polyethylene microplastics (50 and 100 µm) at concentrations from 0.1 to 500 µg/L for up to 24 days, with bioaccumulation found to be dose-dependent and concentrated in the liver and gut. While survival was high (>95%), histopathological damage in liver and intestinal tissue increased significantly with dose.
Eco Toxicological Assessment of Micro Plastic Ingestion in Freshwater Fishes: A Case Study on Bioaccumulation and Histopathological Alterations
Researchers assessed microplastic accumulation in three freshwater fish species (tilapia, rohu, catla) from a major river system, finding microplastics in gastrointestinal tracts and associated histopathological damage in gills, liver, and kidneys.
Histopathological Examination of Zebrafish Organs Under the Exposure to Microplastics
This study conducted histopathological examination of zebrafish organs following microplastic exposure, assessing tissue-level damage in multiple organ systems. The work is part of a broader Kazakhstani research program studying the effects of anthropogenic pollutants on commercial fish species.
Effects of microplastics in freshwater fishes health and the implications for human health
This review examines how microplastics affect the health of freshwater fish, which are a major protein source for billions of people. Fish ingest microplastics that accumulate in their guts, gills, and tissues, leading to inflammation, oxidative stress, and disrupted growth. Since microplastics in fish tissue can transfer to humans through the food chain, this is relevant to both ecosystem and human health.
Dietary administration of PVC and PE microplastics produces histological damage, oxidative stress and immunoregulation in European sea bass (Dicentrarchus labrax L.)
Researchers fed European sea bass diets containing PVC and polyethylene microplastics for three weeks and examined the effects on their organs and immune systems. They found significant tissue damage in the liver and intestine, along with signs of oxidative stress and altered immune function. The study suggests that even short-to-medium-term microplastic ingestion can compromise fish health, with longer exposures potentially leading to irreversible organ damage.
Deleterious Effects of Polypropylene Microplastic Ingestion in Nile Tilapia (Oreochromis niloticus)
Researchers fed Nile tilapia daily doses of polypropylene microplastics for 30 days and observed significant health effects including changes in blood cell counts, altered gut bacteria, and tissue damage to the intestines and liver. The higher dose group showed more pronounced effects, including elevated inflammatory markers and signs of oxidative stress. The study provides evidence that chronic ingestion of microplastics commonly found in aquatic environments can cause meaningful harm to a widely consumed fish species.
Toxic effects of microplastics on freshwater fish (Channa argus): mechanisms of inflammation, apoptosis, and autophagy
Freshwater snakehead fish exposed to polystyrene microplastics for four weeks developed inflammation, cell death, and tissue damage in their liver, intestines, kidneys, and gills. The damage worsened with higher microplastic concentrations and involved disruption of the fish's antioxidant defenses and immune system. Since snakehead is a commonly consumed fish in Asia, these findings raise questions about whether microplastics in aquaculture environments could affect the safety of fish as human food.
Polystyrene microplastics exposure in freshwater fish, Labeo rohita: evaluation of physiology and histopathology
Researchers fed freshwater fish varying levels of polystyrene microplastics for 90 days and found dose-dependent damage to blood health, growth, and organ tissues. Higher microplastic concentrations caused more severe harm to the liver, kidneys, gills, and intestines. The study highlights that microplastics in freshwater systems can accumulate in fish and cause significant health problems, raising concerns about food safety for communities that rely on freshwater fish.
Intestinal alterations in European sea bass Dicentrarchus labrax (Linnaeus, 1758) exposed to microplastics: Preliminary results
Researchers fed European sea bass diets containing virgin and pollutant-contaminated PVC microplastic pellets for 90 days and examined the effects on their intestines. They found moderate tissue damage and increased mucus cell activity in fish exposed to both types of microplastics, with pollutant-laden particles causing the most pronounced changes. The study provides early evidence that chronic microplastic ingestion can alter gut health in commercially important fish species.
Microplastic exposure causes organ damage in Puntius sophore
Researchers sampled a freshwater ecosystem in Tamil Nadu, India, and exposed the local fish Puntius sophore to collected microplastics at sub-lethal concentrations, then assessed organ damage. Microplastic exposure caused histological damage in gills, liver, and kidney of this freshwater sentinel species, with oxidative stress biomarkers elevated in all three organs.
Species-Specific Effects of Microplastics On Juvenile Fishes
This study found that polystyrene microplastics cause intestinal inflammation and tissue damage in three fish species with different feeding habits, with effects varying by fish diet type, plastic size, and exposure duration. The findings show that microplastic-induced gut harm in fish is species-specific, complicating risk assessments for wild fish populations.
Exploring the detrimental effects of microplastics on Asian seabass (Lates calcarifer) fingerlings survival and health
Researchers exposed Asian seabass fingerlings to different concentrations of polyethylene microplastics in their diet and observed significant increases in mortality. Tissue analysis revealed serious liver damage including hepatocyte degeneration and inflammation in fish fed microplastic-contaminated diets. The study provides evidence that dietary microplastic exposure poses a direct threat to the survival and health of farmed fish species.
Understanding the links between micro/nanoplastics-induced gut microbes dysbiosis and potential diseases in fish: A review
This review examines how microplastics and nanoplastics accumulate in fish intestines and disrupt their gut bacteria, potentially leading to inflammation, immune problems, and metabolic diseases. The disrupted gut microbiome can weaken the intestinal barrier, allowing harmful substances to enter the fish's body. Since fish are a major protein source for billions of people, understanding how microplastics damage fish gut health is important for assessing risks to human food safety.
Effect of virgin low density polyethylene microplastic ingestion on intestinal histopathology and microbiota of gilthead sea bream
Researchers fed gilthead sea bream diets containing virgin low-density polyethylene microplastics of two different size ranges for 30 days to assess intestinal effects. They found that microplastic ingestion caused histopathological damage to the intestinal lining and altered the composition of gut microbiota. The study suggests that even virgin microplastics without adsorbed contaminants can disrupt fish intestinal health and microbial balance.
Histomorphological Damage in the Small Intestine of Wami Tilapia (Oreochromis urolepis) (Norman, 1922) Exposed to Microplastics Remain Long after Depuration
Researchers found that histomorphological damage in the small intestine of Wami tilapia larvae persisted even after 60 days of depuration following 65 days of polyethylene microplastic exposure, suggesting long-lasting gut tissue harm from microplastic ingestion.
Microplastics in aquaculture - Potential impacts on inflammatory processes in Nile tilapia
Researchers fed Nile tilapia a diet containing a mixture of four common microplastics and found that the particles triggered inflammatory responses in both adult and juvenile fish. The microplastics activated immune pathways and caused tissue changes in the gut and other organs, with juveniles being more sensitive. Since tilapia is one of the most widely farmed fish in the world, these findings raise concerns about the safety of farm-raised fish exposed to microplastic-contaminated water.
Drenched in microplastic environment: Physiological and metabolic disruptions in fish
This literature review synthesized studies on the physiological and metabolic disruptions microplastics cause in fish, finding impacts across multiple organ systems including the liver, gut, gills, and reproductive organs depending on particle type and exposure duration.
Polystyrene Microplastics Exposure: An Insight into Multiple Organ Histological Alterations, Oxidative Stress and Neurotoxicity in Javanese Medaka Fish (Oryzias javanicus Bleeker, 1854)
Researchers exposed Javanese medaka fish to polystyrene microplastics for 21 days and observed tissue damage across multiple organs including the intestine, liver, and kidney. The study also found increased intestinal oxidative stress and permeability, along with neurotoxicity in the brain characterized by lipid damage and inhibition of the enzyme acetylcholinesterase.
Effects Of Microplastics On Fish Physiology
This review examines how microplastic exposure affects fish physiology, covering accumulation patterns in different tissues, effects on organ function including liver and gill damage, antioxidant responses, and potential reproductive health consequences from both solo and combined contaminant exposures.
Hazards of microplastics exposure to liver function in fishes: A systematic review and meta-analysis
This meta-analysis found that microplastic exposure significantly impairs fish liver function, elevating key liver enzymes (AST, ALT, ALP, LDH) and triggering oxidative stress markers in liver tissue. The toxicological mechanisms include inflammation, apoptosis, and metabolic disruption, raising concerns about the health of fish populations in microplastic-contaminated waters and the safety of fish as a human food source.
Nanoplastics are bioaccumulated in fish liver and muscle and cause DNA damage after a chronic exposure
Researchers chronically exposed fish to nanoplastics and, for the first time, quantified nanoplastic accumulation in liver and muscle tissue. They found that nanoplastics bioaccumulated in these organs and caused DNA damage in the exposed fish. The study provides important evidence that long-term nanoplastic exposure can lead to measurable tissue contamination and genetic harm in aquatic organisms.
Microplastic ingestion cause intestinal lesions in the intertidal fish Girella laevifrons
Researchers fed juvenile intertidal fish diets containing different concentrations of polystyrene microplastics for 45 days and examined intestinal tissue damage. The study found that microplastic ingestion caused dose-dependent intestinal lesions, including increased immune cell infiltration, tissue swelling, and loss of both crypt and villi cells. The severity of tissue damage increased with higher microplastic concentrations, suggesting physical abrasion as a key mechanism of intestinal injury.