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61,005 resultsShowing papers similar to The impact of combined exposure to triphenyltin and microplastics on the oxidative stress, energy metabolism, and digestive function of common carp (Cyprinus carpio)
ClearCombined effect of microplastic and triphenyltin: Insights from the gut-brain axis
Researchers investigated the individual and combined toxicity of microplastics and triphenyltin, an organotin compound, in common carp by examining effects along the gut-brain axis. The study found that co-exposure to microplastics and triphenyltin produced combined toxic effects on the gut microbiome and brain function, suggesting that microplastics may enhance the toxicity of other environmental pollutants through their ability to adsorb contaminants.
Single and combined effects of microplastics and cadmium on juvenile grass carp (Ctenopharyngodon idellus)
Researchers found that combined exposure to polystyrene microplastics and cadmium in juvenile grass carp caused greater physiological stress than either pollutant alone, with microplastics enhancing cadmium accumulation and intensifying oxidative stress and immune responses.
Combined effects of microplastics and benzo[a]pyrene on Asian sea bass Lates calcarifer growth and expression of functional genes
Researchers exposed juvenile Asian sea bass to polyethylene microplastics and the carcinogen benzo[a]pyrene, both individually and in combination, over 56 days. They found that co-exposure caused more severe effects on growth and gene expression related to immune function and stress response than either contaminant alone. The study highlights that microplastics may worsen the toxic effects of chemical pollutants already present in marine environments.
Evaluation of single and combined effects of cadmium and micro-plastic particles on biochemical and immunological parameters of common carp (Cyprinus carpio)
Researchers investigated the individual and combined toxicity of cadmium and polystyrene microplastics on common carp over 30 days. They found that co-exposure to both pollutants produced more severe effects on liver enzymes, immune function, and biochemical markers than either pollutant alone. The study suggests that microplastics can enhance the toxicity of heavy metals in aquatic organisms, raising concerns about the combined impact of these co-occurring contaminants.
Unlocking the combined impact of microplastics and emerging contaminants on fish: A review and meta-analysis
This meta-analysis found that combined exposure to microplastics and emerging contaminants adversely affects fish reproduction, development, and neurotoxicity beyond what either pollutant causes alone. Microplastics with higher adsorption capacities led to more severe outcomes by concentrating and delivering co-contaminants, though exposure duration did not correlate with oxidative stress levels.
Microplastics increase mercury bioconcentration in gills and bioaccumulation in the liver, and cause oxidative stress and damage in Dicentrarchus labrax juveniles
Researchers exposed juvenile European sea bass to mercury, microplastics, and their mixture for 96 hours and found that microplastics increased mercury bioconcentration in gills and bioaccumulation in the liver. The combination of microplastics and mercury also caused greater oxidative stress and lipid damage than either contaminant alone, suggesting microplastics may amplify the toxicity of co-occurring pollutants.
Individual and combined effects of microplastics and diphenyl phthalate as plastic additives on male goldfish: A biochemical and physiological investigation
Male goldfish exposed to both microplastics and the plasticizer chemical DPP (diphenyl phthalate) together showed significant liver damage, disrupted fat and sugar metabolism, and hormonal imbalances including decreased testosterone and increased estrogen. The combined exposure was more harmful than either pollutant alone, demonstrating how microplastics and their chemical additives can work together to disrupt the endocrine system.
Multi‐Biomarkers' Responses in Gills of Oreochromis niloticus Exposed to Glyphosate and Polyethylene Microplastic, Isolated and in Mixture
Researchers exposed tilapia fish to polyethylene microplastics and the herbicide glyphosate, both alone and in combination, and examined gill tissue for signs of damage. They found that the mixture of both contaminants caused more severe oxidative stress and tissue damage than either pollutant alone. The study suggests that microplastics and agricultural chemicals may interact in waterways to amplify harmful effects on fish health.
Combined exposure of emamectin benzoate and microplastics induces tight junction disorder, immune disorder and inflammation in carp midgut via lysosome/ROS/ferroptosis pathway
This study found that when carp were exposed to both the pesticide emamectin benzoate and microplastics together, the damage to their gut lining, immune system, and inflammation levels was significantly worse than exposure to either pollutant alone. The findings suggest that microplastics may amplify the harmful effects of pesticides in aquatic food sources, which could have implications for human health through seafood consumption.
Evaluation of Detoxification‐Related Gene Expression, Oxidative Stress Biomarkers, and Blood Biochemical Parameters in Common Carp ( Cyprinus carpio ) Co‐Exposed to Polyethylene Microplastics and Deltamethrin
Researchers investigated whether polyethylene microplastics worsen the toxic effects of the insecticide deltamethrin in juvenile common carp over a 30-day exposure. The study found that co-exposure to microplastics and deltamethrin affected detoxification-related gene expression, oxidative stress biomarkers, and blood biochemistry, suggesting that microplastics can modify the bioavailability and toxicity of co-occurring pesticides in fish.
Histopathological damage and stress‐ and immune‐related genes' expression in the intestine of common carp, Cyprinus carpio exposed to copper and polyvinyl chloride microparticle
Researchers examined the combined effects of copper and PVC microplastics on common carp intestines, finding that co-exposure caused greater histopathological damage and altered stress- and immune-related gene expression compared to either contaminant alone.
Physiological response and oxidative stress of grass carp (Ctenopharyngodon idellus) under single and combined toxicity of polystyrene microplastics and cadmium
Researchers examined the individual and combined toxicity of polystyrene microplastics and cadmium on grass carp. The study found that fish mortality increased with higher concentrations and that the presence of microplastics elevated cadmium concentration effects, with combined exposure inducing greater oxidative stress than either contaminant alone.
Biochemical effects of polypropylene microplastics on red tilapia (Oreochromis niloticus) after individual and combined exposure with boron
Researchers exposed red tilapia to polypropylene microplastics alone and combined with boron, finding that the mixture disrupted key brain and liver enzymes more severely than either pollutant alone. The study shows that microplastics can amplify the toxic effects of other environmental contaminants in fish.
Neurobehavioral toxicity induced by combined exposure of micro/nanoplastics and triphenyltin in marine medaka (Oryzias melastigma)
When marine medaka fish were exposed to both nanoplastics and the toxic chemical triphenyltin together, they showed much worse nerve and behavioral damage than from either pollutant alone. The combined exposure significantly reduced the fish's swimming ability and disrupted neural gene expression, with smaller nanoplastics causing more severe effects than larger microplastics. This highlights that real-world conditions, where microplastics coexist with other pollutants, may produce amplified toxic effects on the nervous system.
Combined impacts of microplastics and cadmium on the liver function, immune response, and intestinal microbiota of crucian carp (Carassius carassius)
Researchers exposed crucian carp to microplastics and cadmium, both alone and together, and found the combination caused more severe liver damage and immune disruption than either pollutant alone. Co-exposure also significantly altered the fish's gut bacteria after 21 days. This is concerning because microplastics and heavy metals frequently co-occur in polluted waterways, potentially amplifying harm to aquatic life.
Toxic effects of polyethylene microplastics on transcriptional changes, biochemical response, and oxidative stress in common carp (Cyprinus carpio)
Researchers exposed common carp to varying concentrations of polyethylene microplastics and assessed biochemical, oxidative, and gene expression changes. The study found that microplastic exposure caused significant oxidative stress, altered liver enzyme activity, and modified the expression of stress-related genes in a dose-dependent manner.
Combined effects of polystyrene microplastics and copper on antioxidant capacity, immune response and intestinal microbiota of Nile tilapia (Oreochromis niloticus)
Researchers examined the combined effects of polystyrene microplastics and copper on Nile tilapia and found that co-exposure increased copper accumulation in the liver and caused tissue damage in multiple organs. High concentrations of both contaminants together triggered oxidative stress, inflammation, and shifts in intestinal microbial communities. The study suggests that microplastics can worsen the toxic effects of heavy metals on freshwater fish.
Interactive Effects of Microplastics and Tetracycline on Bioaccumulation and Biochemical Status in Jian Carp (Cyprinus carpio var. Jian)
Combined exposure of Jian carp to microplastics and tetracycline affected bioaccumulation of both contaminants and altered intestinal enzyme activity and immune gene expression more than single exposures, demonstrating interactive effects between emerging pollutants in freshwater fish.
Multigenerational effects of combined exposure of triphenyltin and micro/nanoplastics on marine medaka (Oryzias melastigma): From molecular levels to behavioral response
This study exposed marine medaka fish to a combination of micro/nanoplastics and triphenyltin, a toxic chemical used in paints and plastics. The pollutants caused oxidative stress, hormone imbalances, and behavioral changes that carried over to the next generation of fish. The findings show that microplastics combined with other environmental pollutants can cause harm that gets passed down to offspring, raising concerns about long-term effects on marine food webs.
Combined effects of microplastics and copper on oxidative responses in zebrafish (Danio rerio)
Researchers exposed zebrafish (Danio rerio) to microplastics (10 µg/L) and copper (45 µg/L) individually and in combination, finding that fish exposed to both stressors simultaneously exhibited higher oxidative stress across multiple body organs than those exposed to either contaminant alone.
Effects of microplastics on the toxicity of co-existing pollutants to fish: A meta-analysis
Meta-analysis of 1,380 biological endpoints from 55 studies found that microplastics in co-existing pollutant solutions significantly increased toxicity to fish beyond what the pollutants caused alone, particularly elevating immune system damage, metabolic disruption, and oxidative stress. The effect depended on fish life stage and microplastic size, but not on pollutant or polymer type.
The synergetic effects of 4-nonylphenol and polyethylene microplastics in Cyprinus carpio juveniles using blood biomarkers
Researchers found that polyethylene microplastics and the endocrine disruptor 4-nonylphenol produce synergistic toxic effects in juvenile carp, with combined exposure causing greater disruption to blood biomarkers than either contaminant alone.
Single and Combined Effects of Microplastics and Cadmium on the Cadmium Accumulation and Biochemical and Immunity of Channa argus
Researchers investigated the single and combined effects of microplastics and cadmium on juvenile snakehead fish, finding that co-exposure caused greater tissue damage, oxidative stress, and immune disruption than either pollutant alone.
Co-exposure to microplastics and bisphenol A increases viral susceptibility in largemouth bass (Micropterus salmoides) via oxidative stress
Researchers found that juvenile fish exposed to both microplastics and BPA (a chemical found in plastics) together became more susceptible to viral infection, even though neither pollutant alone had that effect. The combination shut down the fish's antioxidant defenses and caused liver cell death, weakening their immune system. This study is important because it shows that common pollutants can interact in unexpected ways, and real-world exposure to multiple contaminants may be more dangerous than lab tests of single substances suggest.