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61,005 resultsShowing papers similar to Microplastics as vectors for microcystin-LR: Enhanced bioavailability, oxidative damage, and immune dysregulation in zebrafish spleen
ClearMicrocystin-LR and polystyrene microplastics jointly lead to hepatic histopathological damage and antioxidant dysfunction in male zebrafish
This study exposed zebrafish to a common water toxin (microcystin-LR) both alone and combined with polystyrene microplastics, and found that the microplastics made the liver damage significantly worse. The microplastics acted as carriers, increasing how much toxin accumulated in the fish liver and amplifying oxidative stress. This is relevant to human health because it shows microplastics can boost the harmful effects of other pollutants commonly found in drinking water sources.
Polystyrene microplastics enhance microcystin-LR-induced cardiovascular toxicity and oxidative stress in zebrafish embryos
Zebrafish embryos exposed to both microplastics and microcystin-LR (a toxin produced by algal blooms) developed significantly worse heart and blood vessel damage than those exposed to the toxin alone. The microplastics amplified oxidative stress and cell death, suggesting that in polluted waterways where both contaminants coexist, the combined health risks may be greater than either one individually.
Polystyrene microplastics enhance the microcystin-LR-induced gonadal damage and reproductive endocrine disruption in zebrafish
Zebrafish exposed to polystyrene microplastics along with microcystin-LR (a common toxin from algae blooms) suffered worse reproductive damage than when exposed to either pollutant alone. The microplastics acted as carriers that increased the amount of toxin accumulating in the fish's reproductive organs. This study demonstrates that microplastics can worsen the effects of other water pollutants by helping toxic chemicals build up in the body.
The presence of polystyrene nanoplastics enhances the MCLR uptake in zebrafish leading to the exacerbation of oxidative liver damage
Researchers found that polystyrene nanoplastics enhanced the uptake of the toxin microcystin-LR in zebrafish liver over three months of co-exposure, exacerbating oxidative damage and cellular swelling compared to microcystin exposure alone.
Influence of microplastics on the accumulation and chronic toxic effects of cadmium in zebrafish (Danio rerio)
Researchers exposed zebrafish to polystyrene microplastics combined with cadmium for three weeks and found that the presence of microplastics significantly increased cadmium accumulation in the liver, gut, and gills. The combined exposure caused greater oxidative damage, tissue inflammation, and disruption of protective gene activity than either pollutant alone. The study demonstrates that microplastics can enhance the toxicity of heavy metals in fish by acting as carriers that increase the body's uptake of harmful substances.
Synergistic reproductive toxicity of microcystin-LR and polystyrene micro/nano-plastics in male zebrafish.
Male zebrafish exposed to both microcystin-LR and polystyrene micro/nano-plastics showed synergistic reproductive toxicity, with co-exposure more severely impairing sperm quality, testicular structure, and reproductive hormones than either contaminant alone. The study underscored the ecological risk posed by the co-occurrence of cyanotoxins and microplastics in aquatic environments.
Combined effects of co-exposure to microcystin-LR and polystyrene microplastics on growth, brain pathology and thyroid hormone homeostasis in adult zebrafish
Researchers exposed zebrafish to microcystin-LR (a toxin from algal blooms) combined with polystyrene microplastics and found that the combination caused significantly worse brain damage and thyroid hormone disruption than either pollutant alone. The microplastics appeared to overwhelm the fish's ability to compensate for the algal toxin, leading to hormone imbalances that could affect growth and development. This is concerning because algal blooms and microplastics frequently occur together in polluted waterways, and their combined effects on the hormone system may be worse than expected.
Toxicological effects of microplastics and phenanthrene to zebrafish (Danio rerio)
Researchers exposed zebrafish to polystyrene microplastics, the pollutant phenanthrene, and a combination of both to assess their toxicity over 24 days. They found that co-exposure amplified oxidative stress, suppressed immune gene expression, and significantly disrupted the gut microbiome compared to either contaminant alone. The study suggests that microplastics can worsen the toxic effects of organic pollutants in aquatic organisms by altering how chemicals accumulate and interact in the body.
Co-exposure to polystyrene microplastics and microcystin-LR aggravated male reproductive toxicity in mice
Researchers found that exposing mice to a combination of polystyrene microplastics and microcystin-LR, a toxin produced by algae, caused more severe damage to male reproductive organs than either pollutant alone. The microplastics increased the amount of the toxin that accumulated in testicular tissue. The study suggests that the interaction between microplastics and other environmental contaminants may amplify reproductive health risks.
Microplastics aggravate the adverse effects of methylmercury than inorganic mercury on zebrafish (Danio rerio)
Researchers exposed zebrafish embryos to polystyrene microplastics combined with two forms of mercury and found that the microplastics significantly increased the accumulation of methylmercury in the fish. The combination of microplastics and methylmercury caused worse developmental abnormalities, delayed hatching, and greater oxidative stress than either pollutant alone. The study suggests that microplastics can act as carriers for toxic metals, amplifying their harmful effects on aquatic organisms.
Potentially Poisonous Plastic Particles: Microplastics as a Vector for Cyanobacterial Toxins Microcystin-LR and Microcystin-LF
Researchers demonstrated for the first time that microplastics can act as vectors for cyanobacterial toxins called microcystins, concentrating the toxins up to 28 times from water onto plastic surfaces. The adsorption process depended on particle size, plastic type, pH, and the specific microcystin variant. The findings raise concerns about microplastics transporting harmful algal toxins through aquatic food webs to higher trophic levels.
The joint effect of parental exposure to microcystin-LR and polystyrene nanoplastics on the growth of zebrafish offspring
Adult zebrafish co-exposed to polystyrene nanoplastics and microcystin-LR (MCLR) for 21 days transferred more MCLR to their offspring than MCLR alone, and the nanoplastic-enhanced MCLR parental load correlated with greater growth inhibition and developmental defects in F1 larvae.
Combined toxic effects of polyethylene microplastics and lambda-cyhalothrin on gut of zebrafish (Danio rerio)
Researchers found that polyethylene microplastics can adsorb the pesticide lambda-cyhalothrin from water and then release it in the guts of zebrafish, worsening its toxic effects. Fish exposed to both microplastics and the pesticide showed greater oxidative stress, immune disruption, and gut microbiome changes than those exposed to the pesticide alone. This demonstrates how microplastics can act as carriers that amplify the toxicity of other environmental pollutants in aquatic organisms consumed by humans.
Polyethylene microplastic exposure and concurrent effect with Aeromonas hydrophila infection on zebrafish
Researchers found that polyethylene microplastic exposure in zebrafish caused oxidative stress, altered antioxidant enzyme activity, and induced intestinal damage, with concurrent Aeromonas hydrophila infection amplifying these toxic effects and increasing mortality rates.
Toxic effects of polystyrene microbeads and benzo[α]pyrene on bioaccumulation, antioxidant response, and cell damage in goldfish Carassius auratus
Researchers exposed goldfish to polystyrene microbeads and benzo[a]pyrene, both individually and in combination, to assess their toxic effects over 120 hours. They found that combined exposure significantly increased oxidative stress, liver damage, and cell death compared to single-compound exposure. The study suggests that microplastics can act as carriers for harmful chemicals, amplifying their toxic impact on aquatic organisms.
Mechanisms of parental co-exposure to polystyrene nanoplastics and microcystin-LR aggravated hatching inhibition of zebrafish offspring
Zebrafish parents co-exposed to microcystin-LR and polystyrene nanoplastics produced offspring with greater MCLR accumulation and more severe hatching inhibition than MCLR alone, with nanoplastics acting as a carrier that enhanced toxin transfer to embryos.
Toxicity of microcystin-LR adsorbed onto microplastics: Impacts on Daphnia magna
Researchers tested how microcystin-LR toxin adsorbed onto polyethylene microplastics affects the freshwater crustacean Daphnia magna compared to free toxin exposure. Microplastic-adsorbed microcystin showed enhanced toxicity versus free toxin at equivalent concentrations, with increased mortality and reduced reproduction, suggesting microplastics can potentiate cyanotoxin hazards in freshwater ecosystems.
Combined effects of microplastics and chemical contaminants on the organ toxicity of zebrafish ( Danio rerio )
Researchers studied the combined effects of microplastics and chemical contaminants like PCBs and methylmercury on zebrafish organs over three weeks of exposure. They found that microplastics carrying adsorbed contaminants produced the most significant effects, particularly on the liver, compared to either microplastics or contaminants alone. The results indicate that microplastics may act as carriers that increase the delivery of harmful chemicals to organisms' tissues.
Toxic effects of co-exposure to polystyrene nanoplastics and arsenic in zebrafish (Danio rerio): Oxidative stress, physiological and biochemical responses
In a zebrafish study, polystyrene nanoplastics made arsenic more toxic by helping the poison build up in the liver, gills, and intestines. The nanoplastics increased cell damage and oxidative stress beyond what arsenic alone would cause. This shows that nanoplastics can act as carriers for other toxic substances in water, potentially making environmental pollutants more dangerous to aquatic life and the food chain.
Comparison of the combined toxicity of polystyrene microplastics and different concentrations of cadmium in zebrafish
Researchers studied the combined toxic effects of polystyrene microplastics and cadmium at different concentrations in zebrafish over a 10-day exposure period. The study found that microplastics could either amplify or reduce cadmium toxicity depending on the metal concentration, affecting survival, growth, and antioxidant responses in complex ways.
Microplastics aggravate the bioaccumulation and toxicity of coexisting contaminants in aquatic organisms: A synergistic health hazard
Researchers conducted a quantitative meta-analysis of 870 endpoints from 40 studies to evaluate whether microplastics increase the bioaccumulation and toxicity of co-occurring contaminants in aquatic organisms. They found that microplastics significantly increased co-contaminant bioaccumulation by 31% and exacerbated toxicity by 18%, with effects manifesting as increased oxidative stress, endocrine disruption, and immunotoxicity. The study confirms that microplastics act as vectors that amplify the hazards of other environmental pollutants.
Toxicity Effects of Microplastics Individually and in Combination the Fish Pathogen Yersinia Ruckeri on the Rainbow Trout (Oncorhynchus Mykiss)
Researchers found that co-exposure of rainbow trout to polystyrene microplastics and the fish pathogen Yersinia ruckeri exacerbated blood biochemical disruptions and hepatic oxidative stress compared to either stressor alone. The results suggest microplastics may act as a predisposing factor that amplifies bacterial infection severity in fish.
Toxic effects of polystyrene microplastics on atrazine in zebrafish: Exogenous toxicity and endogenous mechanism
Researchers found that combining polystyrene microplastics with the common herbicide atrazine was more toxic to zebrafish than either pollutant alone, causing greater liver and gut damage. The combination also degraded water quality by reducing oxygen levels and increasing harmful nitrogen compounds. This is important because microplastics and pesticides frequently co-exist in the environment, meaning their combined effects on aquatic life and food safety may be worse than studies of individual pollutants suggest.
The Role of Synthetic Polymers in the Aquatic Environment and Its Implications in Danio Rerio as a Model Organism
Exposing zebrafish to polystyrene microplastics combined with silver nanoparticles caused significantly more oxidative damage, tissue injury in gills and intestines, and higher mortality than either contaminant alone. The study demonstrates that microplastics can act as carriers that enhance the toxicity of co-pollutants like silver nanoparticles, a combination effect that is highly relevant to understanding real-world aquatic contamination where multiple pollutants co-occur.