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61,005 resultsShowing papers similar to Monitoring of Biochemical Effects of Phenol in the Carp (Cyprinus carpio) Fry
ClearSubacute toxic effects of polyvinyl chloride microplastics (PVC-MPs) in juvenile common carp, Cyprinus carpio (Pisces: Cyprinidae)
Juvenile common carp exposed to polyvinyl chloride microplastics showed dose-dependent toxic effects including tissue damage, oxidative stress, and altered biochemical markers over a subacute exposure period. The results indicate that PVC microplastics are harmful to freshwater fish at ecologically relevant concentrations.
Toxic effects of microplastic (polyethylene) exposure: Bioaccumulation, hematological parameters and antioxidant responses in crucian carp, Carassius carassius
Researchers exposed crucian carp to polyethylene microplastics at various concentrations and found that the particles accumulated in tissues including gills, gut, and liver. The microplastics altered blood cell counts and disrupted the fish's antioxidant defense system in a dose-dependent manner. The study suggests that even common polyethylene microplastics can cause measurable biological harm in freshwater fish.
Polyethylene microplastics increases the tissue damage caused by 4-nonylphenol in the common carp (Cyprinus carpio) juvenile
Researchers found that polyethylene microplastics increased tissue damage caused by the endocrine disruptor 4-nonylphenol in juvenile common carp, with combined exposure producing more severe histopathological changes in gills, liver, and kidneys than individual exposures.
The synergetic effects of 4-nonylphenol and polyethylene microplastics in Cyprinus carpio juveniles using blood biomarkers
Researchers found that polyethylene microplastics and the endocrine disruptor 4-nonylphenol produce synergistic toxic effects in juvenile carp, with combined exposure causing greater disruption to blood biomarkers than either contaminant alone.
The impact of combined exposure to triphenyltin and microplastics on the oxidative stress, energy metabolism, and digestive function of common carp (Cyprinus carpio)
Exposing common carp to triphenyltin and microplastics individually and in combination found that combined exposure caused greater oxidative stress, disrupted energy metabolism more severely, and more strongly impaired digestive enzyme activity than either pollutant alone.
Scruitinizing the Effects of Microplastic (Polyethylene) on Minerals and Liver Enzyme Profile Of Common Carp (Cyprinus carpio)
Researchers exposed common carp to polyethylene microplastics at three concentrations over 7 and 14 days and measured changes in blood minerals and liver enzymes. They found dose- and time-dependent increases in liver damage markers and significant ionic imbalances, including consistent drops in potassium levels. The study suggests that even short-term microplastic exposure can disrupt fish liver function and the ability to maintain proper electrolyte balance.
Alteration in the Antioxidant Enzymes Activities as Potential Biomarkers for Identification of Stress Caused by Afidopyropen Intoxication in Cyprinus Carpio.
This paper is not relevant to microplastics research — it studies oxidative stress biomarkers in common carp (Cyprinus carpio) exposed to afidopyropen, a synthetic insecticide, with no connection to plastic pollution.
The effects of exposure to microplastics on grass carp (Ctenopharyngodon idella) at the physiological, biochemical, and transcriptomic levels
Researchers exposed grass carp to microplastics at two concentrations for 21 days and observed liver damage, inhibited growth, and increased oxidative stress. Transcriptome analysis revealed over 1,500 differentially expressed genes related to immune response, metabolism, and cellular stress pathways. The study suggests that microplastic exposure can trigger broad physiological and molecular disruptions in freshwater fish.
Toxic effects of polyethylene microplastics on transcriptional changes, biochemical response, and oxidative stress in common carp (Cyprinus carpio)
Researchers exposed common carp to varying concentrations of polyethylene microplastics and assessed biochemical, oxidative, and gene expression changes. The study found that microplastic exposure caused significant oxidative stress, altered liver enzyme activity, and modified the expression of stress-related genes in a dose-dependent manner.
Evaluation of Detoxification‐Related Gene Expression, Oxidative Stress Biomarkers, and Blood Biochemical Parameters in Common Carp ( Cyprinus carpio ) Co‐Exposed to Polyethylene Microplastics and Deltamethrin
Researchers investigated whether polyethylene microplastics worsen the toxic effects of the insecticide deltamethrin in juvenile common carp over a 30-day exposure. The study found that co-exposure to microplastics and deltamethrin affected detoxification-related gene expression, oxidative stress biomarkers, and blood biochemistry, suggesting that microplastics can modify the bioavailability and toxicity of co-occurring pesticides in fish.
Metabolic profile changes of zebrafish larvae in the single- and co-exposures of microplastics and phenanthrene
Researchers exposed zebrafish larvae to microplastics and the pollutant phenanthrene, both individually and together, and analyzed changes in their metabolic profiles. They found that combined exposure triggered unique metabolic disruptions not seen with either contaminant alone, particularly in amino acid metabolism pathways. The study provides evidence that microplastics and organic pollutants can interact to produce novel toxic effects in aquatic organisms.
Phenotypic and Gene Expression Alterations in Aquatic Organisms Exposed to Microplastics
This review summarizes research on how microplastics affect aquatic animals at the genetic level, covering changes in hatching, development, and growth. Microplastics, especially when combined with other pollutants, trigger abnormal gene activity in antioxidant and stress-response systems in fish and other water organisms. These genetic disruptions in aquatic life are relevant to human health because affected organisms can enter the food chain through seafood.
Polyvinyl chloride microplastics induce growth inhibition and oxidative stress in Cyprinus carpio var. larvae
Researchers exposed carp larvae to polyvinyl chloride microplastics in their diet for 30 and 60 days at various concentrations. The microplastics significantly inhibited growth and weight gain while causing oxidative stress, altered antioxidant enzyme activities, and changes in gene expression in the liver. Histological examination revealed tissue damage including vacuolation in the liver under higher exposure concentrations.
Effects of Polystyrene Microplastic Exposure on Liver Cell Damage, Oxidative Stress, and Gene Expression in Juvenile Crucian Carp (Carassius auratus)
Researchers exposed young crucian carp to polystyrene microplastics at different concentrations and found dose-dependent liver damage, with higher concentrations causing more severe tissue injury and weaker antioxidant defenses. The microplastics disrupted genes involved in detoxification and stress response in liver cells. Since crucian carp is a commonly consumed freshwater fish, these findings raise questions about whether microplastic-contaminated fish could affect the health of people who eat them.
Ecotoxicological Effects of Polystyrene Particles on Cyprinus carpio: A Laboratory Assessment
Researchers exposed common carp (Cyprinus carpio) to polystyrene particles in a controlled laboratory setting, measuring survival, growth, histological damage, and oxidative stress markers over a 30-day period. Exposed fish showed liver and gill tissue damage alongside elevated oxidative stress enzymes, demonstrating that polystyrene microplastics are harmful to freshwater fish at tested concentrations.
Effects of microplastics on the toxicity of co-existing pollutants to fish: A meta-analysis
Meta-analysis of 1,380 biological endpoints from 55 studies found that microplastics in co-existing pollutant solutions significantly increased toxicity to fish beyond what the pollutants caused alone, particularly elevating immune system damage, metabolic disruption, and oxidative stress. The effect depended on fish life stage and microplastic size, but not on pollutant or polymer type.
Virgin microplastics cause toxicity and modulate the impacts of phenanthrene on biomarker responses in African catfish ( Clarias gariepinus )
Researchers investigated the impacts of virgin and phenanthrene-loaded polyethylene microplastic fragments on juvenile African catfish. The study found that even virgin microplastics caused toxicity and altered biomarker responses, and that microplastics modulated the toxic effects of phenanthrene, suggesting microplastics can act as both independent stressors and carriers of chemical contaminants.
Investigation of the impact caused by different sizes of polyethylene plastics (nano, micro, and macro) in common carp juveniles, Cyprinus carpio L., using multi-biomarkers.
Common carp juveniles exposed to polyethylene plastics of three different sizes (nano, micro, and macro) all showed tissue damage, oxidative stress, and immune disruption, with nanoparticles causing the most severe effects. The size-dependent toxicity pattern suggests that as larger environmental plastics break down into smaller particles, their potential to harm fish — and ultimately people who eat them — may increase.
Dose-Dependent Cytotoxicity of Polypropylene Microplastics (PP-MPs) in Two Freshwater Fishes
Researchers fed zebrafish and freshwater perch polypropylene microplastics at low and high doses and measured cellular damage in liver and gill tissues. They found dose-dependent toxicity including DNA damage up to 18-fold higher than controls, lipid peroxidation, and disrupted metabolism of key compounds like tryptophan. The study provides evidence that polypropylene microplastic ingestion causes significant cellular harm to freshwater fish in a dose-dependent manner.
Effects of PS-MPs on growth, immunity, antioxidant capacity and amino acid metabolism in common carp ( Cyprinus carpio )
Researchers exposed common carp (Cyprinus carpio) to polystyrene microplastics at 0, 50, 100, and 200 micrograms per liter for 15 days, finding that higher concentrations significantly reduced weight gain rates and hepatopancreas somatic indices while impairing immunity, antioxidant capacity, and amino acid metabolism. The results demonstrate dose-dependent toxic effects of PS-MPs on fish physiology at environmentally relevant concentrations.
Assessment the effect of exposure to microplastics in Nile Tilapia (Oreochromis niloticus) early juvenile: I. blood biomarkers
Researchers exposed juvenile Nile tilapia to different concentrations of microplastics for 15 days followed by a 15-day recovery period and measured blood biomarkers. They found dose-dependent changes in biochemical and hematological parameters, including elevated liver enzymes, altered blood cell counts, and increased glucose levels. Many of these effects persisted even after the recovery period, suggesting that microplastic exposure can cause lasting physiological stress in young freshwater fish.
Some Behavioural and Physiological Effects of Plastics (Polyethylene) on Fish
Researchers examined behavioral and physiological effects of polyethylene microplastics on fish, finding that plastic exposure disrupted endocrine function, altered behavior, and impaired normal development and reproduction.
Microplastics induce toxic effects in fish: Bioaccumulation, hematological parameters and antioxidant responses
Researchers exposed juvenile fish to polyamide microplastics and found the particles accumulated primarily in the intestine, gills, and liver, causing reduced blood oxygen-carrying capacity, liver stress, and disrupted antioxidant defenses. These findings matter because fish are an important food source for humans, and microplastic accumulation in fish tissues could transfer these contaminants to people through their diet.
From plankton to fish: The multifaceted threat of microplastics in freshwater environments
This review summarizes how microplastics harm freshwater organisms from tiny plankton to fish through oxidative stress, inflammation, DNA damage, gut microbiome disruption, and metabolic disorders. Microplastics often combine with other pollutants in water, making their toxic effects even worse. Since freshwater systems are a major pathway for microplastics entering oceans and our food supply, understanding these effects is critical for protecting both ecosystems and human health.