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20 resultsShowing papers similar to Polystyrene nanoplastics induce lipid metabolism disorder and alter fatty acid composition in the hepatopancreas of Pacific whiteleg shrimp (Litopenaeus vannamei)
ClearTissue accumulation of polystyrene microplastics causes oxidative stress, hepatopancreatic injury and metabolome alterations in Litopenaeus vannamei
Researchers found that polystyrene microplastics accumulated in shrimp organs, especially the liver-like hepatopancreas, causing growth problems, abnormal swimming, and oxidative stress. Higher microplastic concentrations led to greater tissue damage and disrupted key metabolic pathways including sugar, fat, and amino acid processing. The study expands our understanding of how microplastics affect commercially important seafood species.
Polystyrene nanoplastics exposure alters muscle amino acid composition and nutritional quality of Pacific whiteleg shrimp (Litopenaeus vannamei)
Researchers exposed Pacific whiteleg shrimp to polystyrene nanoplastics at various concentrations for 28 days and measured changes in muscle nutritional quality. They found that higher nanoplastic concentrations reduced growth rates, lowered protein content, and altered the amino acid composition of the shrimp's edible muscle tissue. The study suggests that nanoplastic pollution in aquaculture environments could diminish the nutritional value of farmed seafood.
Effects of nanoplastic exposure on the growth performance and molecular characterization of growth-associated genes in juvenile Macrobrachium nipponense
Researchers found that polystyrene nanoplastic exposure impaired growth in juvenile shrimp by damaging the hepatopancreas, disrupting digestive enzyme activity, and altering growth-related gene expression at concentrations above 10 mg/L.
Exposure to polystyrene nanoplastics induces apoptosis, autophagy, histopathological damage, and intestinal microbiota dysbiosis of the Pacific whiteleg shrimp (Litopenaeus vannamei)
Exposing Pacific white shrimp to nanoplastics caused intestinal damage, cell death, disrupted immune function, and increased the abundance of harmful gut bacteria. Higher concentrations of nanoplastics led to more severe effects, including visible damage to the intestinal lining and formation of autophagosomes (cellular waste structures). These findings add to growing evidence that nanoplastic contamination in seafood farming can compromise the health of organisms that many people eat.
Toxicological effects of polystyrene nanoplastics on marine organisms
Researchers exposed Pacific white shrimp to polystyrene nanoplastics at various concentrations and measured immune, antioxidant, and tissue responses after seven days. They found that nanoplastic exposure disrupted immune function, increased oxidative stress, and caused tissue damage, particularly in the hepatopancreas and gills. The study adds to growing evidence that nanoplastics can harm the health of commercially important marine species.
Oxidative effects of consuming microplastics in different tissues of white shrimp Litopenaeus vannamei
Researchers fed white shrimp diets containing polystyrene microplastics and found the particles accumulated in gills, muscles, and the hepatopancreas. The microplastics triggered oxidative stress, DNA damage, and lipid damage in multiple tissues, along with visible tissue abnormalities including edema and immune cell infiltration. The study demonstrates that dietary microplastic exposure can cause widespread oxidative harm across different organ systems in commercially important shellfish.
Effects of polystyrene nanoplastics on hormonal regulation and glucose metabolism of Pacific whiteleg shrimp (Litopenaeus vannamei)
High concentrations of polystyrene nanoplastics inhibited serum hormone levels, glucose metabolism enzyme activity, and related gene expression in Pacific white-leg shrimp (Litopenaeus vannamei). The study found nanoplastic exposure negatively affects glucose metabolites by inhibiting the pentose phosphate pathway, disrupting energy metabolism in an economically important aquaculture species.
Dietary lipid supplementation alleviated the impacts of polystyrene nanoplastic exposure in Litopenaeus vannamei
Researchers demonstrated that increasing dietary lipid levels from 3% to 9% partially counteracts polystyrene nanoplastic toxicity in Pacific white shrimp, improving survival, weight gain, and lipid metabolism gene expression — the first study to show nutritional intervention can mitigate nanoplastic harm in aquaculture.
Effects of nanoplastics on the gut microbiota of Pacific white shrimp Litopenaeus vannamei
Researchers fed polystyrene nanoplastics to Pacific white shrimp and found significant damage to their gut bacteria, intestinal structure, and immune system. The nanoplastics increased harmful bacteria like Vibrio while reducing beneficial species, and visibly damaged the intestinal lining. Since shrimp are widely consumed seafood, these gut health disruptions raise questions about how nanoplastic contamination in farmed and wild shrimp could affect both the animals and the people who eat them.
Transcriptomic analysis following polystyrene nanoplastic stress in the Pacific white shrimp, Litopenaeus vannamei
Researchers used transcriptomic analysis to study how polystyrene nanoplastics affect gene expression in Pacific white shrimp. They found that nanoplastic exposure activated lysosome pathways and disrupted genes involved in immune response, protein processing, and metabolism. The study provides molecular-level evidence that nanoplastics can interfere with multiple biological systems in commercially important shrimp species.
Accumulation and damage of polyethylene-microplastics to the digestive system of juvenile Litopenaeus vannamei shrimp exposed through feed
Researchers fed juvenile Pacific white shrimp diets containing polyethylene microplastics at varying concentrations for 28 days. While survival and growth were not affected, microplastics accumulated in the digestive system and caused tissue damage to the hepatopancreas and intestine, suggesting that even without visible growth effects, microplastic ingestion can cause internal harm to commercially important crustacean species.
Dietary polystyrene nanoplastics exposure alters liver lipid metabolism and muscle nutritional quality in carnivorous marine fish large yellow croaker (Larimichthys crocea)
Researchers fed polystyrene nanoplastics to large yellow croaker fish for 21 days and found that the particles accumulated in liver cells and disrupted lipid metabolism. The nanoplastics caused excessive fat buildup in the liver and altered the fatty acid composition and texture of fish muscle tissue. The study suggests that nanoplastic contamination in seafood could affect both fish health and the nutritional quality of fish consumed by humans.
Transcriptomic analysis reveals nanoplastics-induced apoptosis, autophagy and immune response in Litopenaeus vannamei
Shrimp exposed to polystyrene nanoplastics for 28 days showed dose-dependent damage to their immune systems, including increased cell death, tissue destruction in the liver-like organ, and disrupted antioxidant defenses. At high concentrations, the nanoplastics overwhelmed the shrimp's ability to fight off threats. Since shrimp are an important food source, these findings raise concerns about the quality and safety of seafood from nanoplastic-contaminated waters.
Effect of polyethylene microplastics on oxidative stress and histopathology damages in Litopenaeus vannamei
Researchers injected fluorescent polyethylene microspheres into Pacific white shrimp (Litopenaeus vannamei) and found that microplastic exposure increased oxidative stress markers and caused histopathological damage to hepatopancreas and gill tissue, even at relatively low concentrations.
Gradual effects of gradient concentrations of polystyrene nanoplastics on metabolic processes of the razor clams
Researchers exposed razor clams to a gradient of polystyrene nanoplastic concentrations and used metabolomics to track effects, finding that even low concentrations disrupted energy metabolism and amino acid pathways, with effects becoming more severe as concentration increased.
Long-Term Exposure to Polystyrene Nanoplastics Impairs the Liver Health of Medaka
Researchers found that three months of exposure to polystyrene nanoplastics caused significant liver damage in medaka fish, including oxidative stress, immune disruption, and altered gene expression related to lipid metabolism and detoxification pathways.
Microplastic-Contaminated Feed Interferes with Antioxidant Enzyme and Lysozyme Gene Expression of Pacific White Shrimp (Litopenaeus vannamei) Leading to Hepatopancreas Damage and Increased Mortality
Researchers fed Pacific white shrimp diets contaminated with high-density polyethylene microplastics and observed dose-dependent immune suppression and organ damage. The microplastics disrupted the expression of antioxidant enzyme and lysozyme genes and caused significant histopathological changes in the hepatopancreas. The study demonstrates that dietary microplastic exposure can compromise the immune defenses of commercially important crustaceans, potentially increasing their susceptibility to disease.
Impacts of polyhydroxybutyrate (PHB) microplastic exposure on physiology and metabolic profiles of Litopenaeus vannamei
Researchers exposed Pacific white shrimp to biodegradable PHB microplastics and found that while short-term exposure up to 100 mg/L showed no acute toxicity, long-term feeding with higher concentrations led to reduced survival, slower growth, and changes in metabolic pathways. The metabolic disruptions included alterations in amino acid and lipid metabolism, suggesting the shrimp's bodies were under significant stress. The findings indicate that even biodegradable plastics can have meaningful negative effects on marine aquaculture species over time.
Size-dependent toxicological effects of polystyrene microplastics in the shrimp Litopenaeus vannamei using a histomorphology, microbiome, and metabolic approach
Researchers exposed white leg shrimp to polystyrene microplastics of four different sizes and found that toxicity was strongly size-dependent, with smaller particles causing greater tissue damage to guts and gills. Smaller microplastics were also more readily ingested and bioavailable to the shrimp. However, larger particles triggered different responses in gut microbiome diversity and metabolic pathways, indicating that microplastic size influences the type and severity of biological effects.
Nanoplastic contamination: Impact on zebrafish liver metabolism and implications for aquatic environmental health
Zebrafish exposed to polystyrene nanoparticles for 28 days showed significant disruptions in liver metabolism, including altered fat processing, signs of inflammation, oxidative stress, and DNA damage. Notably, at lower doses the liver's detox enzymes appeared to break down the nanoplastics themselves, while higher doses overwhelmed these defenses and caused more severe injury.