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61,005 resultsShowing papers similar to Scruitinizing the Effects of Microplastic (Polyethylene) on Minerals and Liver Enzyme Profile Of Common Carp (Cyprinus carpio)
ClearToxic effects of polyethylene microplastics on transcriptional changes, biochemical response, and oxidative stress in common carp (Cyprinus carpio)
Researchers exposed common carp to varying concentrations of polyethylene microplastics and assessed biochemical, oxidative, and gene expression changes. The study found that microplastic exposure caused significant oxidative stress, altered liver enzyme activity, and modified the expression of stress-related genes in a dose-dependent manner.
[Effects of Microplastic Exposure on Crucian Growth, Liver Damage, and Gut Microbiome Composition].
Researchers exposed crucian carp to varying concentrations of polyethylene microplastics in feed for 30 days and found that low-concentration exposure increased body weight while higher concentrations caused liver damage and altered gut microbiome composition, suggesting dose-dependent effects.
Effects of Polystyrene Microplastic Exposure on Liver Cell Damage, Oxidative Stress, and Gene Expression in Juvenile Crucian Carp (Carassius auratus)
Researchers exposed young crucian carp to polystyrene microplastics at different concentrations and found dose-dependent liver damage, with higher concentrations causing more severe tissue injury and weaker antioxidant defenses. The microplastics disrupted genes involved in detoxification and stress response in liver cells. Since crucian carp is a commonly consumed freshwater fish, these findings raise questions about whether microplastic-contaminated fish could affect the health of people who eat them.
Toxic effects of microplastic (polyethylene) exposure: Bioaccumulation, hematological parameters and antioxidant responses in crucian carp, Carassius carassius
Researchers exposed crucian carp to polyethylene microplastics at various concentrations and found that the particles accumulated in tissues including gills, gut, and liver. The microplastics altered blood cell counts and disrupted the fish's antioxidant defense system in a dose-dependent manner. The study suggests that even common polyethylene microplastics can cause measurable biological harm in freshwater fish.
The effects of exposure to microplastics on grass carp (Ctenopharyngodon idella) at the physiological, biochemical, and transcriptomic levels
Researchers exposed grass carp to microplastics at two concentrations for 21 days and observed liver damage, inhibited growth, and increased oxidative stress. Transcriptome analysis revealed over 1,500 differentially expressed genes related to immune response, metabolism, and cellular stress pathways. The study suggests that microplastic exposure can trigger broad physiological and molecular disruptions in freshwater fish.
Deleterious Effects of Polypropylene Microplastic Ingestion in Nile Tilapia (Oreochromis niloticus)
Researchers fed Nile tilapia daily doses of polypropylene microplastics for 30 days and observed significant health effects including changes in blood cell counts, altered gut bacteria, and tissue damage to the intestines and liver. The higher dose group showed more pronounced effects, including elevated inflammatory markers and signs of oxidative stress. The study provides evidence that chronic ingestion of microplastics commonly found in aquatic environments can cause meaningful harm to a widely consumed fish species.
Liver Metabolic Dysregulation Induced by Polypropylene Nano- and Microplastics in Nile Tilapia Using Internal Extractive Electrospray Ionization Mass Spectrometry
Researchers exposed Nile tilapia fish to polypropylene nano- and microplastics and analyzed changes in liver metabolism using advanced mass spectrometry. They identified 46 metabolites that were significantly altered, including phospholipids, amino acids, and energy-related compounds, indicating disrupted liver function. The study suggests that polypropylene plastic particles can cause measurable metabolic disturbances in fish even without obvious visible harm.
Hazards of microplastics exposure to liver function in fishes: A systematic review and meta-analysis
This meta-analysis found that microplastic exposure significantly impairs fish liver function, elevating key liver enzymes (AST, ALT, ALP, LDH) and triggering oxidative stress markers in liver tissue. The toxicological mechanisms include inflammation, apoptosis, and metabolic disruption, raising concerns about the health of fish populations in microplastic-contaminated waters and the safety of fish as a human food source.
Biochemical, Genotoxic and Histological Implications of Polypropylene Microplastics on Freshwater Fish Oreochromis mossambicus: An Aquatic Eco-Toxicological Assessment
Researchers fed polypropylene microplastics to freshwater tilapia and found they caused oxidative stress, DNA damage, and liver tissue deterioration, with more severe effects after 14 days compared to acute 96-hour exposure. The microplastics disrupted antioxidant enzyme systems and neurotransmitter activity in the fish. The study demonstrates that prolonged microplastic ingestion poses a significant ecological threat to freshwater fish species.
Effects of secondary polyethylene microplastic exposure on crucian (Carassius carassius) growth, liver damage, and gut microbiome composition
Researchers exposed crucian carp to secondary polyethylene microplastics derived from agricultural film for 30 days to assess effects on growth, liver health, and gut microbiome. Low and medium concentrations of microplastics actually promoted fish growth by altering gut bacteria, but higher concentrations caused liver damage and disrupted the gut microbiome. The study suggests that microplastic exposure effects on freshwater fish are dose-dependent and can alter both organ health and microbial balance.
Ecotoxicological Effects of Polystyrene Particles on Cyprinus carpio: A Laboratory Assessment
Researchers exposed common carp (Cyprinus carpio) to polystyrene particles in a controlled laboratory setting, measuring survival, growth, histological damage, and oxidative stress markers over a 30-day period. Exposed fish showed liver and gill tissue damage alongside elevated oxidative stress enzymes, demonstrating that polystyrene microplastics are harmful to freshwater fish at tested concentrations.
Hepatic multi-level responses to polyethylene microplastics in Lateolabrax maculatus: Insights from transcriptomics, antioxidant enzyme activity, and histopathology
Researchers exposed spotted sea bass to diets containing polyethylene microplastics for 45 days and found multiple levels of liver damage, including altered gene expression, reduced antioxidant enzyme activity, and visible tissue changes. A subsequent depuration period showed partial but incomplete recovery. The study suggests that chronic microplastic exposure through contaminated feed may pose significant risks to liver health in commercially farmed fish species.
Exploring the detrimental effects of microplastics on Asian seabass (Lates calcarifer) fingerlings survival and health
Researchers exposed Asian seabass fingerlings to different concentrations of polyethylene microplastics in their diet and observed significant increases in mortality. Tissue analysis revealed serious liver damage including hepatocyte degeneration and inflammation in fish fed microplastic-contaminated diets. The study provides evidence that dietary microplastic exposure poses a direct threat to the survival and health of farmed fish species.
Microplastics (Polystyrene) Exposure Induces Metabolic Changes in the Liver of Rare Minnow (Gobiocypris rarus)
Researchers exposed rare minnow fish to polystyrene microplastics and observed significant metabolic changes in the liver after four weeks. The microplastics caused cellular and tissue alterations along with shifts in metabolic pathways related to energy production and lipid metabolism. The study suggests that chronic microplastic exposure can disrupt normal liver function in freshwater fish even at sublethal concentrations.
Exposure to polypropylene microplastics via diet and water induces oxidative stress in Cyprinus carpio
Researchers fed carp fish polypropylene microplastics through both food and water and found that exposure caused oxidative stress in the liver, gills, and intestines. The damage was dose-dependent, with higher microplastic concentrations causing more harm to the fish's antioxidant defense systems. Since carp is a widely consumed fish, these findings raise questions about whether microplastics in aquaculture could affect the safety of fish as human food.
Monitoring of Biochemical Effects of Phenol in the Carp (Cyprinus carpio) Fry
Researchers exposed juvenile carp to phenol — a common industrial pollutant found in wastewater — at different concentrations and measured its effects on protein, lipid, and energy stores in the fish. Higher phenol concentrations caused significant biochemical disruption in the carp. While not specifically about microplastics, phenol is often a co-contaminant with plastic-associated chemicals in polluted water bodies.
Microplastic Bioaccumulation and its Systemic Effects in Labeo rohita: From Cellular Damage to Behavioural Disruption
Rohu carp (Labeo rohita) exposed to polyethylene microplastics at up to 5 mg/L for 60 days accumulated particles in gills, liver, and intestines with dose-dependent cellular damage, behavioral disruption, and partial recovery after 30 days in clean water.
Subacute toxic effects of polyvinyl chloride microplastics (PVC-MPs) in juvenile common carp, Cyprinus carpio (Pisces: Cyprinidae)
Juvenile common carp exposed to polyvinyl chloride microplastics showed dose-dependent toxic effects including tissue damage, oxidative stress, and altered biochemical markers over a subacute exposure period. The results indicate that PVC microplastics are harmful to freshwater fish at ecologically relevant concentrations.
Assessment the effect of exposure to microplastics in Nile Tilapia (Oreochromis niloticus) early juvenile: I. blood biomarkers
Researchers exposed juvenile Nile tilapia to different concentrations of microplastics for 15 days followed by a 15-day recovery period and measured blood biomarkers. They found dose-dependent changes in biochemical and hematological parameters, including elevated liver enzymes, altered blood cell counts, and increased glucose levels. Many of these effects persisted even after the recovery period, suggesting that microplastic exposure can cause lasting physiological stress in young freshwater fish.
Effects of Polyvinyl Chloride Microplastics on the Growth Rate, Liver Enzyme, and Serum Metabolites of Cirrhinus mrigala
Scientists found that plastic particles in water seriously harm fish by damaging their liver and slowing their growth. Since we eat fish and plastic pollution affects the entire food chain, this research suggests that microplastics could eventually impact human health too. The more plastic particles in the water, the sicker the fish became, which is concerning because plastic pollution in our waterways keeps getting worse.
Dose-Dependent Cytotoxicity of Polypropylene Microplastics (PP-MPs) in Two Freshwater Fishes
Researchers fed zebrafish and freshwater perch polypropylene microplastics at low and high doses and measured cellular damage in liver and gill tissues. They found dose-dependent toxicity including DNA damage up to 18-fold higher than controls, lipid peroxidation, and disrupted metabolism of key compounds like tryptophan. The study provides evidence that polypropylene microplastic ingestion causes significant cellular harm to freshwater fish in a dose-dependent manner.
Microplastics accumulation in gut and revealing their impacts on nutritional quality and health of freshwater carp, Catla catla
Researchers fed common carp (Catla catla) diets containing increasing concentrations of polylactic acid microplastics (PLA-MPs) — a type of biodegradable plastic — for 90 days, finding that at higher levels the fish experienced stunted growth, reduced nutrient absorption, altered blood cell counts, and intestinal tissue damage. The study demonstrates that even "biodegradable" microplastics can accumulate in fish and significantly harm their health.
Hepatic transcriptomic and histopathological responses of common carp, Cyprinus carpio, to copper and microplastic exposure
Researchers investigated how PVC microplastics interact with copper exposure in common carp over 14 days. They found that the microplastic particles acted as a vector for copper, increasing its accumulation in the liver and worsening tissue damage beyond what either pollutant caused alone. The study suggests that microplastics can amplify the toxic effects of heavy metals in freshwater fish.
Chronic exposure to high-density polyethylene microplastic through feeding alters the nutrient metabolism of juvenile yellow perch (Perca flavescens)
Researchers fed juvenile yellow perch diets containing varying levels of high-density polyethylene microplastics for nine weeks. While no mortality or growth effects were observed, fish exposed to the highest microplastic levels showed decreased body protein and ash content, enlarged liver cells with increased glycogen storage, reduced liver lipid, and significant intestinal cell damage. The study suggests that chronic microplastic ingestion can alter nutrient metabolism and cause organ-level damage in fish even without affecting survival or growth.