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A. A heat map of toxicity endpoints is generated based on the ADMETlab 3.0 platform.
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This supplementary figure presents toxicity endpoint heat maps for four common microplastic polymers (PE, PP, PVC, PS) generated via computational toxicology, supporting a study linking microplastic molecular targets to allergic rhinitis disease genes.
Red, yellow, and green represent high risk (>0.7), medium risk (0.3–0.7), and low risk (<0.3), respectively. The toxicity scores of each polymer are numerically marked to show the relative risk level. B. 2D structures of polyethylene, polypropylene, polyvinyl chloride, and polystyrene. C.Microplastic-target network diagram. Red squares represent microplastics and blue squares represent corresponding targets.
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Intersection of microplastic toxicity targets and differentially expressed genes in allergic rhinitis.
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Network analysis identified a set of genes that are both targeted by common microplastics (PE, PP, PVC, PS) and differentially expressed in allergic rhinitis, providing a molecular framework for investigating how microplastic exposure may contribute to nasal allergy pathogenesis.
Workflow of the analysis.
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This workflow figure illustrates the analytical pipeline used in a network toxicology study examining the intersection of microplastic molecular targets with allergic rhinitis gene expression data, from database retrieval through enrichment analysis and network construction.
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This figure supplement presents bioinformatics results from a study identifying crossover genes between microplastic toxicity targets and allergic rhinitis differentially expressed genes, including volcano plots, Venn diagrams, and pathway enrichment analyses.
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This dataset and analysis examines the overlap between microplastic toxicity targets and differentially expressed genes in allergic rhinitis, identifying shared molecular pathways that may link microplastic exposure to the development or worsening of allergic airway disease.
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