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61,005 resultsShowing papers similar to Butylparaben-Loaded Aged Polystyrene Nanoplastics Amplify Its Toxicity in Microcystis aeruginosa via Quorum Sensing Suppression and Enhanced Microcystin-LR Release
ClearButylparaben-LoadedAged Polystyrene NanoplasticsAmplify Its Toxicity in Microcystis aeruginosa via Quorum Sensing Suppression and Enhanced Microcystin-LR Release
Researchers found that aged polystyrene nanoplastics adsorbed more butylparaben (a common preservative) than pristine NPs and that butylparaben-loaded aged NPs amplified toxicity to Microcystis aeruginosa by suppressing quorum sensing and increasing the release of the harmful cyanotoxin microcystin-LR. The results highlight how pollutant loading on aged NPs can compound ecological hazards.
Nanoplastics Promote Microcystin Synthesis and Release from Cyanobacterial Microcystis aeruginosa
Researchers discovered that amino-modified polystyrene nanoplastics promote both the production and release of microcystin, a harmful toxin, from the cyanobacterium Microcystis aeruginosa. The nanoplastics inhibited photosynthesis, induced oxidative stress, and damaged cell membranes, which enhanced toxin synthesis and extracellular release. The findings suggest that nanoplastic pollution in freshwater ecosystems could worsen the threat of harmful algal blooms to aquatic ecology and human health.
Nanoplastics promote microcystin synthesis and release from cyanobacterial Microcystis aeruginosa.
Researchers showed that amino-modified polystyrene nanoplastics (PS-NH2) stimulate microcystin synthesis and release in the bloom-forming cyanobacterium Microcystis aeruginosa by inhibiting photosystem II and increasing membrane permeability. This is the first direct evidence linking nanoplastics to enhanced cyanotoxin production in freshwater blooms.
Microcystins-Loaded Aged Nanoplastics Provoke a Metabolic Shift in Human Liver Cells
Researchers found that aged polystyrene nanoplastics can adsorb significantly more microcystin toxins than pristine nanoplastics, and the toxin-loaded particles caused greater harm to human liver cells. The combined exposure triggered metabolic energy disruption, oxidative damage, and stress in cellular machinery. The study suggests that environmentally weathered nanoplastics may pose amplified health risks by carrying higher loads of harmful waterborne toxins.
Micro- and nanoplastic stress intensifies Microcystis aeruginosa physiology and toxin risks under environmentally relevant water chemistry conditions
Researchers exposed the cyanobacterium Microcystis aeruginosa to environmentally relevant concentrations of micro- and nanoplastics, finding both significantly enhanced algal biomass and microcystin toxin production, with nanoplastics additionally promoting extracellular toxin release.
Nitrogen Forms Regulate the Response of Microcystis aeruginosa to Nanoplastics at Environmentally Relevant Nitrogen Concentrations
Researchers found that nanoplastics significantly inhibited the growth of a common blue-green algae species and increased its production of microcystin, a toxin harmful to humans. The type of nitrogen available in the water changed how severely the nanoplastics affected the algae, with nitrate conditions causing the worst growth inhibition. This matters because nanoplastic pollution could increase toxic algal blooms in lakes and reservoirs used for drinking water.
Toxic effects and metabolic response mechanisms of amino-modified polystyrene nanoplastics and arsenic on Microcystis aeruginosa
Researchers investigated the combined effects of amine-modified polystyrene nanoplastics and arsenic on a common freshwater cyanobacterium. They found that co-exposure intensified cellular stress, disrupted metabolic processes, and promoted the release of harmful toxins beyond what either pollutant caused individually. The findings reveal previously unrecognized risks to freshwater ecosystems when nanoplastics interact with heavy metal contaminants.
Mechanistic study on the increase of Microcystin-LR synthesis and release in Microcystis aeruginosa by amino-modified nano-plastics.
This study examined how amino-modified nanoplastics increase production and release of the toxin Microcystin-LR in the cyanobacterium Microcystis aeruginosa, revealing the cellular and gene-expression mechanisms behind this enhancement. The findings highlight how nanoplastic pollution can amplify harmful algal bloom toxicity.
Aging process does not necessarily enhance the toxicity of polystyrene microplastics to Microcystis aeruginosa
Researchers compared the properties and toxicity of pristine versus aged polystyrene microplastics of different sizes on the freshwater cyanobacterium Microcystis aeruginosa. The study found that the aging process does not necessarily increase microplastic toxicity, as aging induced changes in surface properties, functional groups, and zeta potential that could either enhance or reduce toxic effects depending on particle size.
Combined toxicity of nanoplastics and microcystin-LR to sulfate-reducing bacteria and the underlying mechanisms
Researchers exposed freshwater aquaculture microcosms to polyethylene nanoplastics and the algal toxin microcystin-LR, finding that nanoplastics strongly adsorb the toxin and that combined exposure disrupts sulfur cycling bacteria more severely than either contaminant alone, raising ecological concerns for aquaculture water quality.
Toxicity effects of polystyrene nanoplastics and arsenite on Microcystis aeruginosa
Researchers studied how two types of polystyrene nanoplastics with different surface properties interact with arsenic to affect freshwater algae. They found that nanoplastics with a sulfonic acid surface modification caused more severe growth inhibition and metabolic disruption in the algae, while both types reduced arsenic uptake by the organisms. The study highlights that the specific surface chemistry of nanoplastics significantly influences their environmental toxicity.
Aging amplifies the combined toxic effects of polystyrene nanoplastics and norfloxacin on human intestinal cells
Researchers investigated how environmental aging of polystyrene nanoplastics affects their combined toxicity with the antibiotic norfloxacin on human intestinal cells. They found that aged nanoplastics were taken up more readily by cells and significantly amplified the harmful effects of the antibiotic, including increased cell damage. The study suggests that weathered nanoplastics in the environment may pose greater health risks than fresh particles, especially when combined with other contaminants.
Induced aging, structural change, and adsorption behavior modifications of microplastics by microalgae
Researchers found that microalgal biofouling caused more significant aging and surface degradation of microplastics compared to river microbial biofouling over a 30-day period. The study suggests that algae-induced aging substantially enhances the ability of polyethylene and PVC microplastics to adsorb organic pollutants like bisphenol analogues, increasing their potential to transport contaminants in the environment.
Higher toxicity induced by co-exposure of polystyrene microplastics and chloramphenicol to Microcystis aeruginosa: Experimental study and molecular dynamics simulation
Researchers studied what happens when the antibiotic chloramphenicol and polystyrene microplastics are present together in water containing blue-green algae. The study found that the combined exposure was more toxic to the algae than either pollutant alone, disrupting photosynthesis and gene expression. The findings suggest that microplastics and antibiotics may interact in ways that amplify their harmful effects on aquatic ecosystems.
Extracellular polymers substances towards the toxicity effect of Microcystis flos-aquae under subjected to nanoplastic stress
Researchers studied how nanoplastics affect a common freshwater algae and found that the algae produce protective substances in response, but the plastic particles still significantly inhibited growth and disrupted photosynthesis. This matters because harmful algal blooms and water quality are affected by nanoplastic pollution, with downstream consequences for drinking water safety and aquatic food sources.
Modifying luteolin’s algicidal effect on Microcystis by virgin and diversely-aged polystyrene microplastics: Unveiling novel mechanisms through microalgal adaptive strategies
Polystyrene microplastics at concentrations of 0.5-50 mg/L -- both fresh and aged -- weakened the ability of the natural algicide luteolin to suppress Microcystis cyanobacterial blooms by stimulating the algae to produce more protective exopolymers and form aggregates with the plastic particles.
Responses of Microcystis aeruginosa to polystyrene microplastics: Growth dynamics and implications for water treatment
Researchers studied how polystyrene microplastics affect the harmful freshwater algae Microcystis aeruginosa, which causes toxic algal blooms. They found that while microplastics initially suppressed algae growth, the algae eventually adapted and grew even more, producing higher levels of the dangerous toxin microcystin. The study suggests that microplastic pollution in freshwater could worsen harmful algal blooms and create additional water treatment challenges.
Heteroaggregates of Polystyrene Nanospheres and Organic Matter: Preparation, Characterization and Evaluation of Their Toxicity to Algae in Environmentally Relevant Conditions
Polystyrene nanospheres combined with natural organic matter to form heteroaggregates were found to be more toxic to algae under realistic environmental conditions than pristine nanoplastics, highlighting how environmental transformation of nanoplastics can alter their ecological risk.
Effects of photoaged polystyrene microplastics and nanoplastics on the extracellular aggregation and intracellular accumulation of ZnO nanoparticles to algae
When microplastics weather in the environment under UV sunlight, they become more chemically reactive and change how they interact with other pollutants. This study found that photoaged polystyrene microplastics and nanoplastics had a stronger ability to bind zinc oxide nanoparticles than fresh plastic, and that this enhanced binding altered how the zinc nanoparticles affected green algae — generally reducing zinc uptake into algal cells but increasing overall ecological risk. The findings highlight that the environmental "aging" of microplastics is not merely cosmetic — it fundamentally changes their behavior as carriers of other toxic substances in aquatic ecosystems.
Nanoplastics increase algal absorption and toxicity of Cd through alterations in cell wall structure and composition
Lab experiments showed that polystyrene nanoplastics made freshwater algae more vulnerable to cadmium (a toxic heavy metal) by altering the structure of their cell walls, allowing more cadmium to enter the cells. This matters for human health because nanoplastics in waterways may increase how much toxic metal accumulates in aquatic food chains that eventually reach our plates.
Effects of polystyrene nanoplastics on growth and hemolysin production of microalgae Karlodinium veneficum
Researchers exposed the harmful algal bloom species Karlodinium veneficum to polystyrene nanoplastics and found that high concentrations significantly inhibited algal growth and caused oxidative damage to cells. The nanoplastics disrupted cell morphology and weakened photosynthesis and energy metabolism in the algae. Notably, while growth was suppressed, the algae produced more hemolysin toxin, suggesting nanoplastic pollution could make harmful algal blooms more toxic.
Combined effects of microplastics and excess boron on Microcystis aeruginosa
Researchers studied the combined effects of microplastics and excess boron on a common freshwater cyanobacterium (Microcystis aeruginosa). They found that amino-modified polystyrene microplastics were the most harmful, inhibiting growth and worsening boron toxicity, while other surface-modified types actually stimulated growth. The study reveals that the surface chemistry of microplastics plays a key role in how they interact with other pollutants to affect aquatic microorganisms.
Ecological risk analysis and prediction of microplastics' effects on Microcystis aeruginosa in freshwater system: a meta-analysis approach
This meta-analysis found that micro- and nanoplastics can both inhibit and stimulate the growth of Microcystis aeruginosa — a harmful algal bloom cyanobacterium — depending on particle size and degradability. Smaller, degradable plastics tend to promote algal growth, suggesting microplastic pollution could worsen toxic algal blooms in freshwater systems used for drinking water.
Toxicity Effects of Microplastics and Nanoplastics with Cadmium on the Alga Microcystis Aeruginosa
This study tested how microplastics and nanoplastics interact with the heavy metal cadmium to affect the growth of a common freshwater algae, finding that combined exposure was more harmful than either contaminant alone. Nanoplastics adsorbed more cadmium per particle but their smaller size enabled them to penetrate algal cells more easily, with complex effects on cellular toxicity.