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61,005 resultsShowing papers similar to Correctionto “ProteinCorona-Directed CellularRecognition and Uptake of Polyethylene Nanoplastics by Macrophages”
ClearCorrectionto “ProteinCorona-Directed CellularRecognition and Uptake of Polyethylene Nanoplastics by Macrophages”
This entry is a published correction to a prior study on protein corona-directed cellular recognition and uptake of polyethylene nanoplastics by macrophages, noting a correction to previously reported data or methodology.
Correctionto “ProteinCorona-Directed CellularRecognition and Uptake of Polyethylene Nanoplastics by Macrophages”
This entry is a published correction to a prior study on protein corona-directed cellular recognition and uptake of polyethylene nanoplastics by macrophages, noting a correction to previously reported data or methodology.
Correction: Correlation between cellular uptake and cytotoxicity of polystyrene micro/nanoplastics in HeLa cells: A size-dependent matter
This is a correction notice for a previously published study on the correlation between cellular uptake and cytotoxicity of polystyrene micro- and nanoplastics.
Protein Corona-Directed Cellular Recognition and Uptake of Polyethylene Nanoplastics by Macrophages
Scientists discovered that when polyethylene nanoplastics enter the bloodstream, they quickly become coated with blood proteins, and this protein coating determines how immune cells recognize and respond to them. High-density and low-density polyethylene attracted different protein coatings, leading to different immune responses from macrophages. This research helps explain how nanoplastics interact with the immune system once they enter the human body, which is key to understanding their potential health effects.
Correction: Correlation between cellular uptake and cytotoxicity of polystyrene micro/nanoplastics in HeLa cells: A size-dependent matter
This is a correction notice for a published article on polystyrene micro/nanoplastic cytotoxicity and cellular uptake in HeLa cells; it does not report new findings but corrects identified errors in the original publication (DOI: 10.1371/journal.pone.0289473).
Cellular internalization pathways of environmentally exposed microplastic particles: Phagocytosis or macropinocytosis?
Researchers investigated the cellular internalization pathways of environmentally exposed microplastic particles, examining whether phagocytosis or macropinocytosis is the dominant uptake mechanism and how the eco-corona of adsorbed proteins on MP surfaces influences cell-particle interactions.
Compromised Autophagic Effect of Polystyrene Nanoplastics Mediated by Protein Corona Was Recovered after Lysosomal Degradation of Corona
Researchers discovered that when polystyrene nanoplastics enter biological environments, proteins coat their surface forming a protective corona that initially reduces their toxic effects on cells. However, once cells internalize the particles and break down the protein layer in lysosomes, the original toxicity returns, including blocked autophagy and lysosomal damage. The study reveals that protein coronas temporarily mask nanoplastic toxicity rather than permanently neutralizing it.
Cellular internalization pathways of environmentally exposed microplastic particles: Phagocytosis or Macropinocytosis?
Researchers investigated how eco-corona formation on environmentally exposed microplastic particles affects their cellular internalization pathways, examining whether particles coated with biomolecules from freshwater or saltwater are taken up by cells via phagocytosis or macropinocytosis, with findings showing that protein ligands in the eco-corona influence cell membrane receptor interactions and thus uptake mechanisms.
Correction to: No prominent toxicity of polyethylene microplastics observed in neonatal mice following intratracheal instillation to dams during gestational and neonatal period
This is a published correction to an earlier study that found no significant toxicity from polyethylene microplastics in newborn mice exposed through their mothers. The correction addresses a methodological detail in the original paper. The underlying finding — that intratracheal exposure to polyethylene microplastics during pregnancy and nursing caused no prominent toxicity — remains unchanged.
Uptake of extracellular vesicles into immune cells is enhanced by the protein corona
This study found that a protein coating (called a "corona") that forms around nanoparticles in blood actually increases their uptake by human immune cells called monocytes. While this research focused on extracellular vesicles and liposomes rather than plastic particles, the finding is relevant to microplastics research because similar protein coronas form on plastic nanoparticles in the body, potentially influencing how immune cells interact with them.
Cellular internalization pathways of environmentally exposed microplastic particles: Phagocytosis or Macropinocytosis?
Researchers investigated the cellular internalization pathways -- phagocytosis versus macropinocytosis -- by which environmentally exposed microplastic particles enter cells, examining how the eco-corona of biomolecules that forms on particle surfaces in freshwater and saltwater affects cell uptake mechanisms.
Correction: Comprehensive investigation on microplastics from source to sink
This correction clarifies that a prior review paper mischaracterized evidence on microplastic gut translocation, replacing the incorrect statement that most spherical microplastics pass through the gut wall with the accurate interpretation of the cited source.
Correction: Metal–organic framework applications for microplastic remediation: exploring pathways and future potential
This is a published correction notice for a review article on using metal-organic frameworks to remove microplastics — it corrects errors in the original paper and contains no new findings.
Cellular interactions with polystyrene nanoplastics—The role of particle size and protein corona
Researchers investigated how polystyrene nanoplastics interact with mammalian cells, finding that particle size and the protein corona that forms around particles in biological fluids strongly influence cellular uptake and toxicity. Smaller nanoplastics penetrated cell membranes more readily and caused greater disruption, suggesting that the tiniest plastic particles may pose the greatest biological risk.
Correction to: Quantitative analysis of PET microplastics in environmental model samples using quantitative 1H-NMR spectroscopy: validation of an optimized and consistent sample clean-up method
This is a published correction to a prior methodology paper on quantifying PET microplastics in environmental samples using nuclear magnetic resonance spectroscopy. No new findings are presented.
Fate of polystyrene micro- and nanoplastics in zebrafish liver cells: Influence of protein corona on transport, oxidative stress, and glycolipid metabolism
Scientists studied how proteins in biological fluids coat nanoplastic particles (forming a "protein corona") and how this coating changes the way cells take up and process the plastics. The protein coating actually increased how many nanoplastics entered liver cells and made them harder to clear out, suggesting that once nanoplastics enter the bloodstream, the body's own proteins may make the contamination harder to eliminate.
Corrections: Conformational Switching at Cytochrome a During Steady-State Turnover of Cytochrome c Oxidase
The abstract for this entry describes growing global concerns about micro- and nanoplastics and the need for better analytical methods. The title refers to a biochemistry correction notice, indicating a database mismatch.
Macrophage cytoskeletal and immune responses to photoaged and gastrointestinal-transformed polylactic acid micro/nanoplastics with protein corona
Researchers found that UV photoaging and simulated gastrointestinal digestion shrank polylactic acid micro/nanoplastics to one-third their original size and shifted their surface protein corona from lipoprotein-binding to complement and coagulation proteins, yet both pristine and aged particles consistently disrupted the cytoskeleton of human macrophages and triggered formation of macrophage extracellular traps.
Lipid Corona Formation on Micro- and Nanoplastic Particles Modulates Uptake and Toxicity in A549 Cells
Researchers found that lipid corona formation on micro- and nanoplastic particles significantly modulates their cellular uptake and toxicity in human lung cells, suggesting that biological coatings alter how plastic particles interact with human tissues.
Aging of Nanoplastics Significantly Affects Protein Corona Composition Thus Enhancing Macrophage Uptake
Researchers found that when nanoplastics age in the environment through sun exposure, they form a different coating of proteins when they enter the human body compared to fresh nanoplastics. This altered protein coating caused lung immune cells to absorb the aged nanoplastics more readily than new ones. The findings suggest that real-world nanoplastics, which are mostly sun-weathered, may be taken up by the body more aggressively than the fresh particles typically used in lab studies.
Interactions of Micro- and Nanoplastics with Biomolecules: From Public Health to Protein Corona Effect and Beyond
This review summarizes how micro- and nanoplastics interact with biological molecules in the body, including cell membranes and proteins. These particles can cause membranes to thicken and form pores, and they attract a coating of proteins (called a protein corona) that changes how the body responds to them, potentially increasing inflammation, oxidative stress, and disruption of hormone systems.
The in vitro gastrointestinal digestion-associated protein corona of polystyrene nano- and microplastics increases their uptake by human THP-1-derived macrophages
When polystyrene nano- and microplastics pass through simulated gastrointestinal digestion, they acquire a coating of gut proteins — a 'protein corona' — that dramatically increases their uptake by human immune cells (macrophages), boosting internalization of small neutral particles by up to six-fold compared to undigested plastic. The identity of the proteins driving this effect, including clotting factors and apolipoproteins, suggests that realistic dietary exposure conditions substantially change how microplastics interact with the body, and that lab tests using undigested plastics likely underestimate actual cellular uptake.
Unravelling protein corona formation on pristine and leached microplastics
Researchers found that when microplastics encounter proteins in biological fluids, they get coated in a "protein corona" that depends heavily on the plastic's chemical additives, surface area, and how much it has been weathered in the environment. This coating changes how microplastics behave in the body, meaning toxicity studies need to account for these real-world surface changes.
Correction for This Week in PNAS
The abstract for this entry notes growing global concerns about micro- and nanoplastics and the need for better analytical methods. The title is a PNAS correction notice, indicating a database mismatch between title and abstract content.