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Microplastics-perturbed gut microbiota triggered the testicular disorder in male mice: Via fecal microbiota transplantation

Environmental Pollution 2022 71 citations ? Citation count from OpenAlex, updated daily. May differ slightly from the publisher's own count.
Siyue Wen, Yu Zhao, Shanji Liu, Shanji Liu, Hongbin Yuan, Tao You, Hengyi Xu

Summary

Researchers found that polystyrene microplastics disrupted gut microbiota in mice, which in turn triggered testicular damage and reproductive disorders. Through fecal microbiota transplantation experiments, the study suggests that gut bacteria play a critical role in microplastic-induced reproductive toxicity, with inflammatory immune responses driven by bacterial changes being a key mechanism.

Polymers
Models

Microplastics (MPs), an emerging environmental pollutant, have been clarified to induce testicular disorder in mammals. And the current studies have delineated a correlation between gut microbiota and male reproduction. However, it's still unclear whether gut microbiota gets involved in MPs-induced reproductive toxicity. In this work, we constructed a mouse model drinking 5 μm polystyrene-MPs (PS-MPs) at the concentrations of 100 μg/L and 1000 μg/L for 90 days. Evident histological damage, spermatogenetic disorder and hormones synthesis inhibition were observed in PS-MPs exposed mice. With fecal microbiota transplantation (FMT) trial, the recipient mice exhibited gut microbial alteration, and the elevated abundance of Bacteroides and Prevotellaceae_UCG-001 were positively correlated with testicular disorder according to spearman correlation analysis. Mechanistically, increased proportion of pro-inflammatory bacteria may drive translocation of T helper 17 (Th17) cells, resulting in overproduced interleukin (IL)-17 A and downstream inflammatory response in both the mice exposed to PS-MPs and corresponding recipient mice. In summary, our findings revealed the critical role of gut microbiota in PS-MPs-induced reproductive toxicity, and tried to elucidate the underlying mechanism of gut microbial dysregulation-mediated IL-17 A signaling pathway. Furthermore, this study also provides the research basis for gut microbiota-targeted treatment of male infertility in the future.

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