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Article ? AI-assigned paper type based on the abstract. Classification may not be perfect — flag errors using the feedback button. Tier 2 ? Original research — experimental, observational, or case-control study. Direct primary evidence. Nanoplastics Sign in to save

The hazardous impact of true-to-life PET nanoplastics in Drosophila

The Science of The Total Environment 2022 43 citations ? Citation count from OpenAlex, updated daily. May differ slightly from the publisher's own count.
Mohamed Alaraby, Aliro Villacorta, Doaa Abass, Alba Hernández, Ricard Marcos

Summary

Researchers created realistic nanoplastics by sanding commercial PET water bottles and tested their effects on fruit flies (Drosophila melanogaster). They found that these true-to-life nanoplastics were internalized through the digestive tract and distributed throughout the body, causing measurable biological impacts. The study highlights the importance of testing with environmentally relevant plastic particles rather than only laboratory-grade materials to accurately assess health risks.

Polymers
Body Systems

Plastic pollution is a continuously growing problem that can threaten wildlife and human beings. Environmental plastic waste is degraded into small particles termed micro/ nanoplastics (MNPLs) that, due to their small size, can be easily internalized into the exposed organisms, increasing the risks associated with their exposure. To appropriately determine the associated health risk, it is essential to obtain/test representative MNPLs' environmental samples. To such end, we have obtained NPLs resulting from sanding commercial water polyethylene terephthalate (PET) bottles. These true-to-life PETNPLs were extensively characterized, and their potential hazard impacts were explored using Drosophila melanogaster. To highlight the internalization through the digestive tract and the whole body, transmission electron microscopy (TEM) and confocal microscopy were used. In spite of the observed efficient uptake of PETNPLs into symbiotic bacteria, enterocytes, and hemocytes, the exposure failed to reduce flies' survival rates. Nevertheless, PETNPLs exposure disturbed the expression of stress, antioxidant, and DNA repair genes, as well as in those genes involved in the response to physical intestinal damage. Importantly, both oxidative stress and DNA damage induction were markedly increased as a consequence of the exposure to PETNPLs.

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