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Polystyrene microplastics disrupt adrenal steroid synthesis in male mice via mitochondrial dysfunction

Ecotoxicology and Environmental Safety 2024 7 citations ? Citation count from OpenAlex, updated daily. May differ slightly from the publisher's own count. Score: 55 ? 0–100 AI score estimating relevance to the microplastics field. Papers below 30 are filtered from public browse.
Ying Xiong, Zhe Chen, Hanmin Xiang, Yi Liu, Yanlin Wang

Summary

Researchers found that polystyrene microplastics disrupted steroid hormone production in the adrenal glands of male mice by causing mitochondrial dysfunction and oxidative stress. Chronic exposure led to reduced corticosterone levels and increased cell death in adrenal tissue. The study suggests that microplastics may interfere with the body's stress response and hormonal balance through damage to the energy-producing structures within cells.

Polymers
Body Systems
Models
Study Type In vivo

Microplastics have gained significant social attention, as they can enter our bodies through food and drinking water. The adrenal gland is essential for the maintenance of metabolic homeostasis and stress responses. Nevertheless, the effects of microplastics on the steroid synthesis in the adrenal cortex was still unclear. In this study, through both in vivo and in vitro models, we found that polystyrene microplastics (PS-MPs) impaired adrenal steroid synthesis, leading to a reduction in corticosterone levels. In vivo, we further observed that chronic exposure to PS-MPs (0.25, 0.5 and 1 mg/d for 4 weeks) could induce abnormal mitochondrial morphology and functional disruptions of adrenal glands in male mice, along with an imbalance in cellular oxidative stress, manifested as increased level of reactive oxygen species, diminished antioxidant activity (glutathione peroxidase and superoxide dismutase). In vitro, these occurrences coincided with an elevated rate of cell apoptosis observed in adrenocortical cells following exposure to PS-MPs. We proposed that mitochondrial dysfunction not only directly influenced the biosynthetic processes of steroid hormones but also induced cell apoptosis through the initiation of cellular oxidative stress. The latter may represent a common mechanism underlying the multi-organ toxicity induced by PS-MPs in the body. Our findings would provide new insights for the development of more effective environmental protection measures and the reduction of plastic pollution.

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