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Polystyrene nanoplastics induce multi-organ oxidative stress in mice: experimental evidence and a descriptive random forest-based analysis of study-design variables.

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Mice given tiny polystyrene plastic particles showed increasing signs of cell damage in the lung, liver, kidney, brain and testicles as the dose rose, along with lung inflammation and tissue injury. Mice also given the substance N-acetylcysteine had less lung damage. The study also reviewed existing research patterns on this topic.

Polymers
Models
Study Type In vivo

Polystyrene nanoplastics (PS-NPs), ubiquitous in natural environments, readily accumulate in organisms and pose emerging toxicological risks. This study pairs in vivo animal experiments assessing organ-specific oxidative stress responses induced by PS-NPs exposure with a descriptive random forest-based analysis of study-design variables reported in the published literature. Kunming mice were administered varying concentrations of PS-NPs with or without N-acetylcysteine (NAC) to assess oxidative stress across five organs (lung, liver, kidney, brain, and testicles). Dose-dependent increases in ROS and MDA, accompanied by GSH depletion, were observed in the lung, liver, kidney, and brain; testicles ROS and GSH followed similar trends, although MDA plateaued at the high dose without further significant increase. Additionally, pulmonary inflammatory cytokines (e.g., TNF-α, IL-1β, IL-6) and histopathological examination revealed dose-dependent lung injury, which was also significantly alleviated by NAC co-administration. A descriptive literature mining further illustrated the current distribution of study design variables across organ-specific investigations, highlighting the existing research focus and knowledge gaps in PS-NPs toxicology. Collectively, this work demonstrates the complementary value of pairing toxicological experimentation with descriptive literature analysis to decipher nanoplastic health risks and highlights the promise of data-driven analysis for toxicology and environmental health assessment.

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