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61,005 resultsShowing papers similar to Mechanistic study on the increase of Microcystin-LR synthesis and release in Microcystis aeruginosa by amino-modified nano-plastics.
ClearNanoplastics Promote Microcystin Synthesis and Release from Cyanobacterial Microcystis aeruginosa
Researchers discovered that amino-modified polystyrene nanoplastics promote both the production and release of microcystin, a harmful toxin, from the cyanobacterium Microcystis aeruginosa. The nanoplastics inhibited photosynthesis, induced oxidative stress, and damaged cell membranes, which enhanced toxin synthesis and extracellular release. The findings suggest that nanoplastic pollution in freshwater ecosystems could worsen the threat of harmful algal blooms to aquatic ecology and human health.
Nanoplastics promote microcystin synthesis and release from cyanobacterial Microcystis aeruginosa.
Researchers showed that amino-modified polystyrene nanoplastics (PS-NH2) stimulate microcystin synthesis and release in the bloom-forming cyanobacterium Microcystis aeruginosa by inhibiting photosystem II and increasing membrane permeability. This is the first direct evidence linking nanoplastics to enhanced cyanotoxin production in freshwater blooms.
Micro- and nanoplastic stress intensifies Microcystis aeruginosa physiology and toxin risks under environmentally relevant water chemistry conditions
Researchers exposed the cyanobacterium Microcystis aeruginosa to environmentally relevant concentrations of micro- and nanoplastics, finding both significantly enhanced algal biomass and microcystin toxin production, with nanoplastics additionally promoting extracellular toxin release.
The photosynthetic toxicity of nano-polystyrene to Microcystis aeruginosa is influenced by surface modification and light intensity
Researchers found that amino-modified nanoplastics are more toxic to the cyanobacterium Microcystis aeruginosa than unmodified particles, and that high light intensity amplifies this toxicity by generating additional reactive oxygen species — including singlet oxygen and hydroxyl radicals — through interactions between visible light and the particle surface.
Micrometer scale polystyrene plastics of varying concentrations and particle sizes inhibit growth and upregulate microcystin-related gene expression in Microcystis aeruginosa
Researchers found that polystyrene microplastics inhibited the growth of the cyanobacterium Microcystis aeruginosa in a dose- and size-dependent manner, with smaller particles and higher concentrations causing greater growth suppression. Notably, microplastic exposure also upregulated genes related to microcystin production, suggesting that microplastics could potentially increase the toxicity of harmful algal blooms.
Acute effects of three surface-modified nanoplastics against Microcystis aeruginosa: Growth, microcystin production, and mechanisms
Researchers found that surface-modified nanoplastics significantly inhibited growth of the cyanobacterium Microcystis aeruginosa while increasing microcystin production by up to 175%, with positively charged particles causing the strongest effects.
Toxicity mechanism of Nylon microplastics on Microcystis aeruginosa through three pathways: Photosynthesis, oxidative stress and energy metabolism
Researchers investigated how nylon microplastics affect the freshwater cyanobacterium Microcystis aeruginosa and found dose-dependent growth inhibition reaching nearly 48% at the highest concentration. The microplastics disrupted photosynthesis, damaged cell membranes, triggered oxidative stress, and altered the expression of genes involved in energy production and carbon fixation. The study identifies three interconnected pathways through which nylon microplastics harm these important aquatic organisms.
Polymer-specific toxicity of microplastics to Microcystis aeruginosa: Growth inhibition, physiological responses, and molecular mechanisms
Researchers exposed the cyanobacterium Microcystis aeruginosa to four polymer types over 12 days and found that all significantly inhibited growth, with PVC causing the greatest inhibition, and identified polymer-specific molecular mechanisms including oxidative stress and photosynthesis disruption.
Nitrogen Forms Regulate the Response of Microcystis aeruginosa to Nanoplastics at Environmentally Relevant Nitrogen Concentrations
Researchers found that nanoplastics significantly inhibited the growth of a common blue-green algae species and increased its production of microcystin, a toxin harmful to humans. The type of nitrogen available in the water changed how severely the nanoplastics affected the algae, with nitrate conditions causing the worst growth inhibition. This matters because nanoplastic pollution could increase toxic algal blooms in lakes and reservoirs used for drinking water.
Ecological risk analysis and prediction of microplastics' effects on Microcystis aeruginosa in freshwater system: a meta-analysis approach
This meta-analysis found that micro- and nanoplastics can both inhibit and stimulate the growth of Microcystis aeruginosa — a harmful algal bloom cyanobacterium — depending on particle size and degradability. Smaller, degradable plastics tend to promote algal growth, suggesting microplastic pollution could worsen toxic algal blooms in freshwater systems used for drinking water.
Combined effects of microplastics and excess boron on Microcystis aeruginosa
Researchers studied the combined effects of microplastics and excess boron on a common freshwater cyanobacterium (Microcystis aeruginosa). They found that amino-modified polystyrene microplastics were the most harmful, inhibiting growth and worsening boron toxicity, while other surface-modified types actually stimulated growth. The study reveals that the surface chemistry of microplastics plays a key role in how they interact with other pollutants to affect aquatic microorganisms.
Post-exposure recovery of Microcystis aeruginosa from nanoplastics stress: metabolic adaptation and damage resilience
Researchers exposed Microcystis aeruginosa cyanobacteria to polystyrene nanoplastics for 15 days, then transferred them to NP-free medium to study post-exposure recovery. Toxicity was concentration-dependent during exposure, and cells showed metabolic changes and only partial recovery after removal, suggesting persistent effects on cyanobacterial physiology.
Effects of polystyrene microplastics on growth, physiological traits of Microcystis aeruginosa and microcystin production and release
Researchers examined how polystyrene microplastics of various sizes affect the growth and toxin production of the harmful algae Microcystis aeruginosa. They found that microplastics inhibited algal growth at low densities, with the smallest particles causing the greatest inhibition, and also disrupted the algae's antioxidant defense system. Notably, microplastic exposure led to a significant increase in the production of the toxin microcystin-LR, raising concerns about how microplastic pollution could worsen harmful algal blooms.
Toxic effects and metabolic response mechanisms of amino-modified polystyrene nanoplastics and arsenic on Microcystis aeruginosa
Researchers investigated the combined effects of amine-modified polystyrene nanoplastics and arsenic on a common freshwater cyanobacterium. They found that co-exposure intensified cellular stress, disrupted metabolic processes, and promoted the release of harmful toxins beyond what either pollutant caused individually. The findings reveal previously unrecognized risks to freshwater ecosystems when nanoplastics interact with heavy metal contaminants.
Combined toxicity of nanoplastics and microcystin-LR to sulfate-reducing bacteria and the underlying mechanisms
Researchers exposed freshwater aquaculture microcosms to polyethylene nanoplastics and the algal toxin microcystin-LR, finding that nanoplastics strongly adsorb the toxin and that combined exposure disrupts sulfur cycling bacteria more severely than either contaminant alone, raising ecological concerns for aquaculture water quality.
Spatio-temporal variation of toxin-producing gene abundance in Microcystis aeruginosa from Poyang Lake
This paper is not relevant to microplastics; it investigates the spatio-temporal variation of toxin-producing gene abundance in the cyanobacterium Microcystis aeruginosa in Poyang Lake, China.
Polystyrene nanoplastics affect growth and microcystin production of Microcystis aeruginosa
Researchers exposed Microcystis aeruginosa to polystyrene nanoplastics across a range of concentrations and tracked effects on growth, cell aggregation, and microcystin production and release throughout the full growth cycle. They found a dose-dependent growth inhibition and increased aggregation at high concentrations, but nanoplastics at 50 mg/L paradoxically stimulated a period of rapid growth, with complex effects on intracellular and extracellular microcystin levels.
Extracellular polymers substances towards the toxicity effect of Microcystis flos-aquae under subjected to nanoplastic stress
Researchers studied how nanoplastics affect a common freshwater algae and found that the algae produce protective substances in response, but the plastic particles still significantly inhibited growth and disrupted photosynthesis. This matters because harmful algal blooms and water quality are affected by nanoplastic pollution, with downstream consequences for drinking water safety and aquatic food sources.
Elucidation of Mechanisms by Which Microplastics (PET) Facilitates the Rapid Growth of Benthic Cyanobacteria and Toxin Production in Aquatic Ecosystems
This review examines how PET microplastics and their chemical leachates may promote the growth of benthic cyanobacteria in aquatic environments. The study suggests that PET-derived compounds can serve as carbon sources or signaling molecules that alter gene expression related to photosynthesis and stress responses, potentially contributing to harmful cyanobacterial blooms in plastic-polluted waters.
Butylparaben-Loaded Aged Polystyrene Nanoplastics Amplify Its Toxicity in Microcystis aeruginosa via Quorum Sensing Suppression and Enhanced Microcystin-LR Release
Researchers found that aged nanoplastics (those weathered by the environment) are much better at absorbing and carrying the chemical butylparaben into harmful algae called Microcystis aeruginosa. The contaminated nanoplastics caused the algae to release more of a dangerous toxin called microcystin-LR, which can contaminate drinking water. This study shows how nanoplastics can amplify the harmful effects of other pollutants in water systems that supply human drinking water.
Fate, abundance and ecological risks of microcystins in aquatic environment: The implication of microplastics
This review explores how microplastics in water can interact with microcystins, highly toxic compounds produced by harmful algal blooms, by adsorbing and transporting them through aquatic environments. The combination poses increased risks to human health because microplastics can carry these dangerous toxins into drinking water sources and through the food chain.
Microcystis aeruginosa's exposure to an antagonism of nanoplastics and MWCNTs: The disorders in cellular and metabolic processes
Researchers examined the combined effects of polystyrene nanoplastics and multi-walled carbon nanotubes on the cyanobacterium Microcystis aeruginosa, discovering antagonistic interactions that disrupted cellular and metabolic processes in this freshwater organism.
Microplastics benefit bacteria colonization and induce microcystin degradation
Polystyrene microplastics in a microcosm experiment facilitated bacterial colonization and promoted the degradation of the cyanobacterial toxin microcystin, with the plastisphere community showing distinct metabolic activity compared to free-living bacteria. The study reveals that microplastic biofilms can unexpectedly accelerate detoxification of co-occurring harmful algal bloom toxins.
Toxicity effects of microplastics and nanoplastics with cadmium on the alga Microcystis aeruginosa
Researchers examined the combined toxicity of microplastics, nanoplastics, and cadmium on the freshwater alga Microcystis aeruginosa. The study found that while cadmium alone was most toxic, the combination of plastics and cadmium produced synergistic harmful effects, with nanoplastics causing greater cadmium release and more severe disruption to algal cell membranes than microplastics.