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61,005 resultsShowing papers similar to Post-exposure recovery of Microcystis aeruginosa from nanoplastics stress: metabolic adaptation and damage resilience
ClearData Sheet 1_Post-exposure recovery of Microcystis aeruginosa from nanoplastics stress: metabolic adaptation and damage resilience.docx
This data supplement reported post-exposure recovery experiments with Microcystis aeruginosa after polystyrene nanoplastic stress. After nanoplastic removal, cyanobacterial cells showed metabolic and physiological recovery, but some toxicity effects persisted, indicating that nanoplastic exposure causes both reversible and lasting changes in algal biology.
Micro- and nanoplastic stress intensifies Microcystis aeruginosa physiology and toxin risks under environmentally relevant water chemistry conditions
Researchers exposed the cyanobacterium Microcystis aeruginosa to environmentally relevant concentrations of micro- and nanoplastics, finding both significantly enhanced algal biomass and microcystin toxin production, with nanoplastics additionally promoting extracellular toxin release.
Nanoplastics Promote Microcystin Synthesis and Release from Cyanobacterial Microcystis aeruginosa
Researchers discovered that amino-modified polystyrene nanoplastics promote both the production and release of microcystin, a harmful toxin, from the cyanobacterium Microcystis aeruginosa. The nanoplastics inhibited photosynthesis, induced oxidative stress, and damaged cell membranes, which enhanced toxin synthesis and extracellular release. The findings suggest that nanoplastic pollution in freshwater ecosystems could worsen the threat of harmful algal blooms to aquatic ecology and human health.
The photosynthetic toxicity of nano-polystyrene to Microcystis aeruginosa is influenced by surface modification and light intensity
Researchers found that amino-modified nanoplastics are more toxic to the cyanobacterium Microcystis aeruginosa than unmodified particles, and that high light intensity amplifies this toxicity by generating additional reactive oxygen species — including singlet oxygen and hydroxyl radicals — through interactions between visible light and the particle surface.
Polystyrene nanoplastics affect growth and microcystin production of Microcystis aeruginosa
Researchers exposed Microcystis aeruginosa to polystyrene nanoplastics across a range of concentrations and tracked effects on growth, cell aggregation, and microcystin production and release throughout the full growth cycle. They found a dose-dependent growth inhibition and increased aggregation at high concentrations, but nanoplastics at 50 mg/L paradoxically stimulated a period of rapid growth, with complex effects on intracellular and extracellular microcystin levels.
Nanoplastics promote microcystin synthesis and release from cyanobacterial Microcystis aeruginosa.
Researchers showed that amino-modified polystyrene nanoplastics (PS-NH2) stimulate microcystin synthesis and release in the bloom-forming cyanobacterium Microcystis aeruginosa by inhibiting photosystem II and increasing membrane permeability. This is the first direct evidence linking nanoplastics to enhanced cyanotoxin production in freshwater blooms.
Microcystis aeruginosa copes with toxic effects of micro/nano-plastics with varying particle sizes through different self-regulatory mechanisms
Researchers exposed the freshwater cyanobacterium Microcystis aeruginosa to polystyrene particles of three different sizes ranging from nanoscale to microscale. All particle sizes harmed the algae, but they triggered different cellular defense mechanisms depending on their size, with nanoparticles causing the most severe damage. The findings reveal that particle size is a key factor in determining how microplastics affect aquatic microorganisms.
Micrometer scale polystyrene plastics of varying concentrations and particle sizes inhibit growth and upregulate microcystin-related gene expression in Microcystis aeruginosa
Researchers found that polystyrene microplastics inhibited the growth of the cyanobacterium Microcystis aeruginosa in a dose- and size-dependent manner, with smaller particles and higher concentrations causing greater growth suppression. Notably, microplastic exposure also upregulated genes related to microcystin production, suggesting that microplastics could potentially increase the toxicity of harmful algal blooms.
Microcystis aeruginosa's exposure to an antagonism of nanoplastics and MWCNTs: The disorders in cellular and metabolic processes
Researchers examined the combined effects of polystyrene nanoplastics and multi-walled carbon nanotubes on the cyanobacterium Microcystis aeruginosa, discovering antagonistic interactions that disrupted cellular and metabolic processes in this freshwater organism.
Polymer-specific toxicity of microplastics to Microcystis aeruginosa: Growth inhibition, physiological responses, and molecular mechanisms
Researchers exposed the cyanobacterium Microcystis aeruginosa to four polymer types over 12 days and found that all significantly inhibited growth, with PVC causing the greatest inhibition, and identified polymer-specific molecular mechanisms including oxidative stress and photosynthesis disruption.
Aging process does not necessarily enhance the toxicity of polystyrene microplastics to Microcystis aeruginosa
Researchers compared the properties and toxicity of pristine versus aged polystyrene microplastics of different sizes on the freshwater cyanobacterium Microcystis aeruginosa. The study found that the aging process does not necessarily increase microplastic toxicity, as aging induced changes in surface properties, functional groups, and zeta potential that could either enhance or reduce toxic effects depending on particle size.
Polystyrene nanoplastics trigger changes in cell surface properties of freshwater and marine cyanobacteria
Polystyrene nanoplastics altered cell surface properties—including charge, hydrophobicity, and extracellular polymeric substance composition—in both freshwater and marine cyanobacteria without affecting growth or structure, suggesting cyanobacteria employ adaptive surface remodeling strategies to resist nanoplastic stress.
Persistence of algal toxicity induced by polystyrene nanoplastics at environmentally relevant concentrations
Researchers studied whether the harmful effects of polystyrene nanoplastics on marine algae are temporary or long-lasting. They found that while some damage, like oxidative stress, was reversible after exposure ended, other effects such as increased cell membrane damage persisted. The study suggests that even at low, environmentally realistic concentrations, nanoplastics can cause lasting disruption to algal metabolism and cell function.
Mechanistic study on the increase of Microcystin-LR synthesis and release in Microcystis aeruginosa by amino-modified nano-plastics.
This study examined how amino-modified nanoplastics increase production and release of the toxin Microcystin-LR in the cyanobacterium Microcystis aeruginosa, revealing the cellular and gene-expression mechanisms behind this enhancement. The findings highlight how nanoplastic pollution can amplify harmful algal bloom toxicity.
Responses of Microcystis aeruginosa to polystyrene microplastics: Growth dynamics and implications for water treatment
Researchers studied how polystyrene microplastics affect the harmful freshwater algae Microcystis aeruginosa, which causes toxic algal blooms. They found that while microplastics initially suppressed algae growth, the algae eventually adapted and grew even more, producing higher levels of the dangerous toxin microcystin. The study suggests that microplastic pollution in freshwater could worsen harmful algal blooms and create additional water treatment challenges.
Toxicity mechanism of Nylon microplastics on Microcystis aeruginosa through three pathways: Photosynthesis, oxidative stress and energy metabolism
Researchers investigated how nylon microplastics affect the freshwater cyanobacterium Microcystis aeruginosa and found dose-dependent growth inhibition reaching nearly 48% at the highest concentration. The microplastics disrupted photosynthesis, damaged cell membranes, triggered oxidative stress, and altered the expression of genes involved in energy production and carbon fixation. The study identifies three interconnected pathways through which nylon microplastics harm these important aquatic organisms.
Roles of extracellular polymeric substances on Microcystis aeruginosa exposed to different sizes of polystyrene microplastics
Researchers examined how the cyanobacterium Microcystis aeruginosa responds to different sizes of polystyrene microplastics by producing extracellular polymeric substances. They found that the composition of these protective substances varied depending on particle size, with each size triggering distinct defense mechanisms in the algae. The study reveals that extracellular polymeric substances play a crucial role in mitigating the adverse effects of microplastics on algal growth and photosynthesis.
Toxic effects and metabolic response mechanisms of amino-modified polystyrene nanoplastics and arsenic on Microcystis aeruginosa
Researchers investigated the combined effects of amine-modified polystyrene nanoplastics and arsenic on a common freshwater cyanobacterium. They found that co-exposure intensified cellular stress, disrupted metabolic processes, and promoted the release of harmful toxins beyond what either pollutant caused individually. The findings reveal previously unrecognized risks to freshwater ecosystems when nanoplastics interact with heavy metal contaminants.
Responses of bloom-forming Microcystis aeruginosa to polystyrene microplastics exposure: Growth and photosynthesis
Researchers exposed bloom-forming blue-green algae (Microcystis aeruginosa) to polystyrene microplastics and found a complex pattern: high concentrations (50–100 mg/L) temporarily suppressed growth and photosynthesis in the middle of the experiment, but promoted growth at the beginning and end. This suggests microplastics could worsen harmful algal blooms in the long run, which is concerning because these blooms produce toxins that contaminate drinking water.
Size-dependent toxic effects of polystyrene microplastic exposure on Microcystis aeruginosa growth and microcystin production
Researchers exposed the freshwater cyanobacterium Microcystis aeruginosa to polystyrene microplastics of two sizes and found that particle size significantly influenced the effects. The larger 1-micrometer particles promoted algal growth while aggregating on cell surfaces and inhibiting photosynthesis, whereas 100-nanometer particles stimulated toxin production. The study suggests that microplastic pollution in freshwater may have complex, size-dependent effects on harmful algal blooms and their toxin output.
Acute effects of three surface-modified nanoplastics against Microcystis aeruginosa: Growth, microcystin production, and mechanisms
Researchers found that surface-modified nanoplastics significantly inhibited growth of the cyanobacterium Microcystis aeruginosa while increasing microcystin production by up to 175%, with positively charged particles causing the strongest effects.
Changes of the physicochemical properties of extracellular polymeric substances (EPS) from Microcystis aeruginosa in response to microplastics
This study examined how microplastics affect the extracellular polymeric substances produced by the common freshwater cyanobacterium Microcystis aeruginosa, which plays a role in harmful algal blooms. Researchers found that microplastic exposure altered the composition and structure of these substances over time. The findings suggest that microplastics could influence how cyanobacteria aggregate and form blooms, with potential implications for water quality management.
Unravelling the toxicity mechanisms of polystyrene nanoplastics on physiological and transcriptomic responses of the marine dinoflagellate Alexandrium minutum
Researchers exposed the toxic marine dinoflagellate Alexandrium minutum to polystyrene nanoplastics at concentrations from 0.1 to 50 mg/L and measured physiological responses and toxin production. NP exposure inhibited growth and photosynthesis, altered gene expression, and changed the profile of paralytic shellfish toxins produced by the alga.
Microplastics benefit bacteria colonization and induce microcystin degradation
Polystyrene microplastics in a microcosm experiment facilitated bacterial colonization and promoted the degradation of the cyanobacterial toxin microcystin, with the plastisphere community showing distinct metabolic activity compared to free-living bacteria. The study reveals that microplastic biofilms can unexpectedly accelerate detoxification of co-occurring harmful algal bloom toxins.