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61,005 resultsShowing papers similar to Growth inhibition, toxin production and oxidative stress caused by three microplastics in Microcystis aeruginosa
ClearPolymer-specific toxicity of microplastics to Microcystis aeruginosa: Growth inhibition, physiological responses, and molecular mechanisms
Researchers exposed the cyanobacterium Microcystis aeruginosa to four polymer types over 12 days and found that all significantly inhibited growth, with PVC causing the greatest inhibition, and identified polymer-specific molecular mechanisms including oxidative stress and photosynthesis disruption.
Comparative growth and cellular responses of toxigenic Microcystis exposed to different types of microplastics at various doses
Researchers exposed toxigenic Microcystis cyanobacteria to polyethylene and polyvinyl chloride microplastics at various concentrations to study dose- and time-dependent effects. They found that low microplastic doses initially stimulated growth, while higher doses increasingly inhibited it, with PVC showing stronger effects than polyethylene. The study suggests that microplastic pollution in freshwaters could influence the behavior of harmful algal blooms depending on the type and concentration of plastic present.
Effects of polystyrene microplastics on growth, physiological traits of Microcystis aeruginosa and microcystin production and release
Researchers examined how polystyrene microplastics of various sizes affect the growth and toxin production of the harmful algae Microcystis aeruginosa. They found that microplastics inhibited algal growth at low densities, with the smallest particles causing the greatest inhibition, and also disrupted the algae's antioxidant defense system. Notably, microplastic exposure led to a significant increase in the production of the toxin microcystin-LR, raising concerns about how microplastic pollution could worsen harmful algal blooms.
Micrometer scale polystyrene plastics of varying concentrations and particle sizes inhibit growth and upregulate microcystin-related gene expression in Microcystis aeruginosa
Researchers found that polystyrene microplastics inhibited the growth of the cyanobacterium Microcystis aeruginosa in a dose- and size-dependent manner, with smaller particles and higher concentrations causing greater growth suppression. Notably, microplastic exposure also upregulated genes related to microcystin production, suggesting that microplastics could potentially increase the toxicity of harmful algal blooms.
Size-dependent toxic effects of polystyrene microplastic exposure on Microcystis aeruginosa growth and microcystin production
Researchers exposed the freshwater cyanobacterium Microcystis aeruginosa to polystyrene microplastics of two sizes and found that particle size significantly influenced the effects. The larger 1-micrometer particles promoted algal growth while aggregating on cell surfaces and inhibiting photosynthesis, whereas 100-nanometer particles stimulated toxin production. The study suggests that microplastic pollution in freshwater may have complex, size-dependent effects on harmful algal blooms and their toxin output.
Nanoplastics Promote Microcystin Synthesis and Release from Cyanobacterial Microcystis aeruginosa
Researchers discovered that amino-modified polystyrene nanoplastics promote both the production and release of microcystin, a harmful toxin, from the cyanobacterium Microcystis aeruginosa. The nanoplastics inhibited photosynthesis, induced oxidative stress, and damaged cell membranes, which enhanced toxin synthesis and extracellular release. The findings suggest that nanoplastic pollution in freshwater ecosystems could worsen the threat of harmful algal blooms to aquatic ecology and human health.
Responses of Microcystis aeruginosa to polystyrene microplastics: Growth dynamics and implications for water treatment
Researchers studied how polystyrene microplastics affect the harmful freshwater algae Microcystis aeruginosa, which causes toxic algal blooms. They found that while microplastics initially suppressed algae growth, the algae eventually adapted and grew even more, producing higher levels of the dangerous toxin microcystin. The study suggests that microplastic pollution in freshwater could worsen harmful algal blooms and create additional water treatment challenges.
Toxicity mechanism of Nylon microplastics on Microcystis aeruginosa through three pathways: Photosynthesis, oxidative stress and energy metabolism
Researchers investigated how nylon microplastics affect the freshwater cyanobacterium Microcystis aeruginosa and found dose-dependent growth inhibition reaching nearly 48% at the highest concentration. The microplastics disrupted photosynthesis, damaged cell membranes, triggered oxidative stress, and altered the expression of genes involved in energy production and carbon fixation. The study identifies three interconnected pathways through which nylon microplastics harm these important aquatic organisms.
Responses of bloom-forming Microcystis aeruginosa to polystyrene microplastics exposure: Growth and photosynthesis
Researchers exposed bloom-forming blue-green algae (Microcystis aeruginosa) to polystyrene microplastics and found a complex pattern: high concentrations (50–100 mg/L) temporarily suppressed growth and photosynthesis in the middle of the experiment, but promoted growth at the beginning and end. This suggests microplastics could worsen harmful algal blooms in the long run, which is concerning because these blooms produce toxins that contaminate drinking water.
Micro- and nanoplastic stress intensifies Microcystis aeruginosa physiology and toxin risks under environmentally relevant water chemistry conditions
Researchers exposed the cyanobacterium Microcystis aeruginosa to environmentally relevant concentrations of micro- and nanoplastics, finding both significantly enhanced algal biomass and microcystin toxin production, with nanoplastics additionally promoting extracellular toxin release.
Ecological risk analysis and prediction of microplastics' effects on Microcystis aeruginosa in freshwater system: a meta-analysis approach
This meta-analysis found that micro- and nanoplastics can both inhibit and stimulate the growth of Microcystis aeruginosa — a harmful algal bloom cyanobacterium — depending on particle size and degradability. Smaller, degradable plastics tend to promote algal growth, suggesting microplastic pollution could worsen toxic algal blooms in freshwater systems used for drinking water.
Effects of Polyester Microfibers on the Growth and Toxicity Production of Bloom-Forming Cyanobacterium Microcystis aeruginosa
Green, black, and white polyester microplastic fibers at concentrations of 10-200 mg/L affected the growth, photosynthesis, and toxin production of the bloom-forming cyanobacterium Microcystis aeruginosa in color- and concentration-dependent ways. Black microplastics caused the greatest inhibition of growth while simultaneously altering microcystin production, suggesting MPs could shift the hazard profile of harmful algal blooms.
Microplastic characteristics differentially influence cyanobacterial harmful algal bloom microbial community membership, growth, and toxin production
Researchers investigated how different types of microplastics influence the growth and toxin production of harmful algal blooms in freshwater. They found that certain microplastic characteristics, such as shape and polymer type, significantly affected which microbial species thrived and how much toxin was produced. The study suggests that microplastic pollution may play an underappreciated role in worsening harmful algal blooms in lakes and reservoirs.
Combined effects of microplastics and excess boron on Microcystis aeruginosa
Researchers studied the combined effects of microplastics and excess boron on a common freshwater cyanobacterium (Microcystis aeruginosa). They found that amino-modified polystyrene microplastics were the most harmful, inhibiting growth and worsening boron toxicity, while other surface-modified types actually stimulated growth. The study reveals that the surface chemistry of microplastics plays a key role in how they interact with other pollutants to affect aquatic microorganisms.
Nitrogen Forms Regulate the Response of Microcystis aeruginosa to Nanoplastics at Environmentally Relevant Nitrogen Concentrations
Researchers found that nanoplastics significantly inhibited the growth of a common blue-green algae species and increased its production of microcystin, a toxin harmful to humans. The type of nitrogen available in the water changed how severely the nanoplastics affected the algae, with nitrate conditions causing the worst growth inhibition. This matters because nanoplastic pollution could increase toxic algal blooms in lakes and reservoirs used for drinking water.
Nanoplastics promote microcystin synthesis and release from cyanobacterial Microcystis aeruginosa.
Researchers showed that amino-modified polystyrene nanoplastics (PS-NH2) stimulate microcystin synthesis and release in the bloom-forming cyanobacterium Microcystis aeruginosa by inhibiting photosystem II and increasing membrane permeability. This is the first direct evidence linking nanoplastics to enhanced cyanotoxin production in freshwater blooms.
Single and combined effects of microplastics and lead on the freshwater algae Microcystis aeruginosa
Researchers tested the individual and combined effects of microplastics and lead (Pb) on the growth, photosynthetic pigments, and antioxidant responses of the freshwater cyanobacterium Microcystis aeruginosa. They found that microplastics alone inhibited growth while low-dose Pb promoted it, but their combination altered toxicity outcomes in complex ways depending on concentration, indicating that co-exposure risks in freshwater cannot be predicted from single-contaminant studies.
Effects of micro-sized biodegradable plastics on Microcystis aeruginosa
Researchers tested how micro-sized biodegradable plastics made from polylactic acid and polyhydroxybutyrate affect a common freshwater cyanobacterium. They found that even biodegradable microplastics inhibited the growth and photosynthetic activity of the organism, though to a lesser extent than conventional plastics. The study suggests that switching to biodegradable plastics does not eliminate the risk of microplastic-related harm to aquatic microorganisms.
Effects of combined exposure of PVC and PFOA on the physiology and biochemistry of Microcystis aeruginosa
Researchers examined the combined effects of PVC microplastics and the PFAS chemical PFOA on a common freshwater algae species. They found that the combination inhibited algal growth and promoted the release of microcystin toxins, while also causing physical damage to the cells. The study suggests that the co-presence of microplastics and PFAS in water bodies may create compounding risks for aquatic ecosystems.
Uncovering the potential effect of microplastics on Alexandrium pacificum: From the perspective of cyst formation and toxin production
Microplastics were found to influence the growth and toxin production of Alexandrium (a harmful algal bloom species), with effects depending on plastic type and concentration. This raises concerns that microplastic pollution could alter the frequency or severity of harmful algal blooms in coastal waters.
Toxicity effects of microplastics and nanoplastics with cadmium on the alga Microcystis aeruginosa
Researchers examined the combined toxicity of microplastics, nanoplastics, and cadmium on the freshwater alga Microcystis aeruginosa. The study found that while cadmium alone was most toxic, the combination of plastics and cadmium produced synergistic harmful effects, with nanoplastics causing greater cadmium release and more severe disruption to algal cell membranes than microplastics.
Evaluating physiological responses of microalgae towards environmentally coexisting microplastics: A meta-analysis
A meta-analysis of 52 studies found that microplastics inhibit microalgal growth and photosynthesis and induce oxidative damage, though microalgae can recover over time. Cyanobacteria are more vulnerable than green algae, and the relative size of microplastics to algal cells governs the mechanism of impact, while aged versus pristine microplastics have opposite effects on extracellular polymeric substance and microcystin production.
Microcystis aeruginosa's exposure to an antagonism of nanoplastics and MWCNTs: The disorders in cellular and metabolic processes
Researchers examined the combined effects of polystyrene nanoplastics and multi-walled carbon nanotubes on the cyanobacterium Microcystis aeruginosa, discovering antagonistic interactions that disrupted cellular and metabolic processes in this freshwater organism.
Acute Toxicity Effects of Aged Polyethylene and Polylactic Acid Microplastics on Microcystis aeruginosa: Growth and Oxidative Stress Response
Researchers compared the acute toxicity of aged polyethylene (conventional plastic) and polylactic acid (biodegradable plastic) microplastics on the cyanobacterium Microcystis aeruginosa. Aged PLA microplastics inhibited algal growth more than aged PE, and UV-aged particles were more toxic than heat-aged ones for both plastic types. The study suggests that biodegradable plastics may not be less harmful than conventional plastics once they begin degrading in the environment.