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Exposure of juvenile turbot ("Scophthalmus maximus") to contaminated microplastic diet: oxidative stress biomarkers, erythrocytic nuclear abnormalities and histopathological effects
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Researchers exposed juvenile olive flounder to polyethylene microplastics at concentrations up to 2,500 mg/L for 96 hours and observed significant reductions in blood glucose, cholesterol, calcium, and liver enzyme AST without causing mortality. These findings indicate that acute microplastic exposure induces measurable blood physiological disruptions in fish even at sublethal doses.
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Researchers reviewed the physiological and immunological effects of microplastic exposure in fish, finding that these particles smaller than 5 mm induce a range of biological disruptions including immune modulation, oxidative stress, and systemic health impacts with broader implications for aquatic ecosystem integrity.
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Benni fish exposed to increasing concentrations of polyethylene microplastics showed significant liver enzyme disruption, elevated cholesterol and triglycerides, and clear signs of oxidative damage in their cells. These dose-dependent biochemical changes suggest that microplastic exposure causes systemic physiological stress in fish, raising concerns about the health of species humans consume.
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