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Lipidomic evaluation of single and combined effects of polyethylene microplastics and two polychlorobiphenyl congeners on human hepatoma cells
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Lipidomic analysis of single and combined effects of polyethylene microplastics and polychlorinated biphenyls on human hepatoma cells
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Researchers used lipidomic analysis to study the single and combined effects of polyethylene microplastics and polychlorinated biphenyls on human liver cancer cells. While microplastics alone did not cause significant cell death, the combined exposure with PCBs produced distinct changes in cellular lipid profiles that differed from individual exposures. The study suggests that microplastics may alter how cells respond to co-occurring chemical pollutants through a "Trojan Horse" transport mechanism.
Dataset for the manuscript "Lipidomic analysis of single and combined effects of polyethylene microplastics and polychlorinated biphenyls on human hepatoma cells"
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This dataset contains lipidomics data from human hepatoma cells exposed to polyethylene microplastics and polychlorinated biphenyls, both individually and in combination. The research data support findings that co-exposure to microplastics and PCBs produces distinct lipid profile changes in human liver cells compared to individual exposures.
Dataset for the manuscript "Lipidomic analysis of single and combined effects of polyethylene microplastics and polychlorinated biphenyls on human hepatoma cells"
AI summary Read the abstract
This dataset provides untargeted lipidomics data from human hepatoma cells exposed to polyethylene microplastics, polychlorinated biphenyl congeners, and their combinations. The data support research showing that microplastics and PCBs can individually and jointly alter lipid profiles in human liver cells, offering insights into how these contaminants may affect cellular lipid metabolism.
Disruption of lipid metabolism under plastic siege: Unraveling the toxic mechanisms of microplastics and nanoplastics
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Researchers reviewed how microplastics and nanoplastics disrupt lipid metabolism across multiple animal species, finding they inhibit digestive enzymes, alter gut microbiota, dysregulate fat storage and breakdown, and activate inflammatory signaling pathways — causing both acute and chronic metabolic damage.
The toxicity of polyethylene microplastics on molecular and biochemical parameters in albino mice
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Researchers assessed the molecular and biochemical effects of polyethylene microplastic exposure in albino mice, finding dose-dependent toxicity across multiple organ systems, with measurable alterations in liver enzymes, oxidative stress markers, and blood cell parameters consistent with systemic health impacts.
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