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Article ? AI-assigned paper type based on the abstract. Classification may not be perfect — flag errors using the feedback button. Tier 2 ? Original research — experimental, observational, or case-control study. Direct primary evidence. Detection Methods Environmental Sources Human Health Effects Nanoplastics Remediation Sign in to save

Nanoplastics aggravate the toxicity of arsenic to AGS cells by disrupting ABC transporter and cytoskeleton

Ecotoxicology and Environmental Safety 2021 61 citations ? Citation count from OpenAlex, updated daily. May differ slightly from the publisher's own count. Score: 55 ? 0–100 AI score estimating relevance to the microplastics field. Papers below 30 are filtered from public browse.
Lei He, Chengyong Li Lei He, Lei He, Pei‐Chun Lin, Pei‐Chun Lin, Pei‐Chun Lin, Pei‐Chun Lin, Chengyong Li Pei‐Chun Lin, Lei He, Zifan Lu, Pei‐Chun Lin, Lei He, Lei He, Yitao Guo, Lei He, Zifan Lu, Zifan Lu, Zifan Lu, Lei He, Lei He, Lei He, Chengyong Li Pei‐Chun Lin, Lei He, Lei He, Yitao Guo, Pei‐Chun Lin, Lei He, Lei He, Lei He, Chengyong Li Lei He, Lei He, Zifan Lu, Pei‐Chun Lin, Lei He, Lei He, Lei He, Lei He, Pei‐Chun Lin, Zifan Lu, Zifan Lu, Lei He, Lei He, Lei He, Lei He, Lei He, Xiuchun Liao, Xiuchun Liao, Xiuchun Liao, Xiuchun Liao, Chunxia Zhou, Lei He, Lei He, Lei He, Zifan Lu, Pengzhi Hong, Chengyong Li Shengli Sun, Xueru Chen, Shengli Sun, Liuying He, Lei He, Xueru Chen, Lei He, Lei He, Liuying He, Yitao Guo, Liuying He, Lei He, Lei He, Zifan Lu, Zhong‐Ji Qian, Pengzhi Hong, Pengzhi Hong, Pengzhi Hong, Zhong‐Ji Qian, Pengzhi Hong, Liuying He, Zhong‐Ji Qian, Yitao Guo, Chengyong Li Zhong‐Ji Qian, Chengyong Li Chengyong Li Shengli Sun, Shengli Sun, Shengli Sun, Shengli Sun, Shengli Sun, Pengzhi Hong, Pengzhi Hong, Pengzhi Hong, Zifan Lu, Chengyong Li Shengli Sun, Shengli Sun, Chengyong Li Zhong‐Ji Qian, Shengli Sun, Shengli Sun, Zhong‐Ji Qian, Chengyong Li Chengyong Li Chengyong Li Chengyong Li Chengyong Li Pengzhi Hong, Lei He, Pengzhi Hong, Pengzhi Hong, Shengli Sun, Chengyong Li Chengyong Li Chengyong Li Shengli Sun, Chunxia Zhou, Chengyong Li Lei He, Chengyong Li Shengli Sun, Shengli Sun, Pengzhi Hong, Chengyong Li Chengyong Li Chengyong Li Chengyong Li Chengyong Li Chengyong Li Shengli Sun, Pengzhi Hong, Pengzhi Hong, Pengzhi Hong, Lei He, Chengyong Li Chengyong Li Chengyong Li Chengyong Li Chengyong Li Chengyong Li Shengli Sun, Shengli Sun, Pengzhi Hong, Chengyong Li Chengyong Li Chengyong Li Zhong‐Ji Qian, Chengyong Li Chengyong Li Chengyong Li Pengzhi Hong, Shengli Sun, Shengli Sun, Chengyong Li Chengyong Li Chengyong Li Chengyong Li Chengyong Li

Summary

Researchers investigated how polystyrene nanoplastics of various sizes affect the toxicity of arsenic in human gastric cells. They found that while nanoplastics alone at noncytotoxic concentrations did not harm cells, they significantly enhanced arsenic accumulation and toxicity by disrupting cell membrane integrity, damaging the cytoskeleton, and inhibiting ABC transporter activity. The study highlights that nanoplastics can aggravate the harmful effects of co-occurring environmental contaminants even at concentrations previously considered safe.

Polymers

The coexistence of nanoplastics (NPs) and pollutants such as arsenic (As) has become an unignorable environmental problem. However, there is still a considerable knowledge gap about the impact of NPs and pollutants on human health risks. In this study, the human gastric adenocarcinoma (AGS) cells were used as a model to investigate the toxicity of NPs with different particle sizes and As by MTT assay, western blotting, immunofluorescence and so on. The results showed that 20 nm (8 μg/mL), 50 nm (128 μg/mL), 200 nm (128 μg/mL), 500 nm (128 μg/mL), 1000 nm (128 μg/mL) polystyrene (PS) did not affect cell viability, ROS, intracellular calcium and activate apoptosis pathway in AGS cells. However, noncytotoxic concentration of NPs enhanced the cytotoxicity and intracellular accumulation of As. NPs destroys the fluidity of cell membrane and cytoskeleton, inhibits the activity of ABC transporter, and leads to the accumulation of As in cells. This work highlights that the damage caused by NPs, especially at the level of noncytotoxicity, joint with As cannot be ignored and provides a specific toxicological mechanism of NPs accompanied by exposure to As.

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