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Low-Density Polyethylene Microplastics in the Blood Seems not Induce Alzheimer’s Disease in Wistar Rat
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Researchers investigated whether low-density polyethylene microplastics in the blood trigger Alzheimer's disease in Wistar rats, noting that these particles enter the human body through contaminated food sources including table salt, fish, tea bags, and drinking water.
Microplastic particles <5 mm in the blood pose health problems to humans. One of the entry points for low density polyethylene microplastics in the human blood is through consumption of contaminated food. These low density polyethylene microplastic particles are found in table salt, canned sardines, beer, sea fish, honey, sugar, tea bags, minerals and drinking water. These findings estimated between 37 to as high as billion microplastic particles from those various food products. The number of low density polyethylene microplastic particles that contaminate food and beverages will continue to increase along with the increase in plastic debris in the environment.
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Microplastics detection in freshly harvested human brain tissues of northeast Chinese postmortem donors and their correlation with Alzheimer's disease-related pathological proteins.
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Researchers examined brain tissue from 16 deceased donors in northeast China and found microplastics, including polyethylene, PVC, polystyrene and polypropylene, in all samples. Higher levels of two plastic types were linked to higher amounts of a protein associated with Alzheimer's disease, though this does not prove the plastics caused the protein changes.
The Potential Presence of Microplastics in Daily Food and Beverage Consumption May Lead to Alzheimer's disease in The Thai Population
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This review examines the potential presence of microplastics in everyday food and beverages including seafood, tap water, bottled water, and packaged foods, summarizing current contamination data and dietary exposure estimates. The authors highlight the need for standardized methods to improve comparability across studies.
A novel risk factor for dementia: chronic microplastic exposure
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This review examines emerging evidence that chronic microplastic exposure may be a previously overlooked risk factor for dementia. Microplastics can cross the blood-brain barrier and may promote brain damage through oxidative stress, inflammation, and by accelerating the buildup of amyloid plaques linked to Alzheimer's disease, with studies finding higher microplastic levels in the brains of dementia patients compared to controls.
Membranes and deoxyribonucleic acid of hippocampal neurons damage due to low-density polyethylene microplastics in blood of Wistar rats
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Researchers found that low-density polyethylene microplastics in rat blood caused dose-dependent membrane lipid peroxidation and DNA damage in hippocampal neurons, providing experimental evidence that circulating microplastics can reach and harm brain tissue.
Alzheimer’s disease: the role of extrinsic factors in its development, an investigation of the environmental enigma
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This review examines how environmental contaminants, including microplastics, may contribute to Alzheimer's disease risk. Along with air pollutants, toxic metals, and pesticides, plastics and microplastics are identified as potential risk factors that may promote brain inflammation and neurodegeneration. The review explores the molecular mechanisms behind these effects and suggests strategies to reduce the brain health impacts of environmental contaminants.
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