0
Article Tier 2 Nanoplastics Sign in to save

Study the disassembly of epithelial tight junction under the influence of nanoplastics using an in vitro multicellular model

No summary available — this paper's abstract is not included in the open metadata provided by the publisher. Learn why →

Study Type In vitro

More Papers Like This

Article Tier 2

Nanoplastic-Induced Biological Effects In Vivo and In Vitro: An Overview

AI summary Read the abstract

Researchers reviewed how nanoplastics accumulate in and harm living organisms, finding that particle size, surface charge, exposure dose, and duration all influence toxicity — with smaller, charged particles penetrating cells more readily and chemical additives amplifying harm beyond the plastics themselves.

Article Tier 2

Microplastics and nanoplastics in human health: a comprehensive review of exposure pathways, cellular mechanisms, and toxicological implications

AI summary Read the abstract

Researchers reviewed human exposure pathways to microplastics and nanoplastics through seafood, salt, drinking water, inhalation, and skin contact, examining how these particles cross biological barriers to reach organs and trigger oxidative stress, inflammation, and endocrine disruption with poorly understood long-term consequences.

Article

Toxicological effects of different particle sizes and types of nano-plastics (NPs) on human lung cells: implications for the health risk of airborne microplastic pollution

AI summary Read the abstract

Researchers exposed human A549 alveolar cells to polystyrene and polymethylmethacrylate nanoplastics of 30 and 50 nm and found that smaller size and higher concentration produced stronger cytotoxicity, oxidative stress, inflammation, and apoptosis—with PS nanoparticles more toxic than PMMA—providing in vitro evidence of respiratory health risks from inhaled airborne nanoplastics.

Article

The effects of nanoplastics on toxicity of metals and metal nanoparticles: molecular mechanisms and health effects

AI summary Read the abstract

Researchers reviewed how co-exposure to nanoplastics and heavy metals produces synergistic toxicity across multiple species and cell models, finding that nanoplastics enhance metal bioavailability and amplify oxidative stress, mitochondrial dysfunction, apoptosis, and gut dysbiosis more severely than equivalent microplastic co-exposures.

Article Tier 2

Cellular Uptake, Transport, and Organelle Response After Exposure to Microplastics and Nanoplastics: Current Knowledge and Perspectives for Environmental and Health Risks

AI summary Read the abstract

Microplastics and nanoplastics enter cells through endocytosis and passive diffusion, then travel through organelles including endosomes, autophagosomes, and lysosomes, ultimately causing toxicity in mitochondria, endoplasmic reticulum, nucleus, and cytoskeleton. Understanding these cellular uptake pathways and organelle-level damage mechanisms is critical for accurately assessing the human health risks of chronic microplastic exposure.

Research digests by email

When a large batch of papers lands in the Atlas, we read through it and send a short write-up of what stood out.

Email me about

Share this paper