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Article ? AI-assigned paper type based on the abstract. Classification may not be perfect — flag errors using the feedback button. Tier 2 ? Original research — experimental, observational, or case-control study. Direct primary evidence. Environmental Sources Human Health Effects Sign in to save

Polystyrene Microplastics Exacerbate Candida albicans Infection Ability In Vitro and In Vivo

International Journal of Molecular Sciences 2023 9 citations ? Citation count from OpenAlex, updated daily. May differ slightly from the publisher's own count. Score: 50 ? 0–100 AI score estimating relevance to the microplastics field. Papers below 30 are filtered from public browse.
Rosa Carotenuto, Rosa Carotenuto, Emilia Galdiero, Angela Maione, Antonia Feola, Rosa Carotenuto, Mariangela Norcia, Rosa Carotenuto, Marica Sinoca, Marica Sinoca, Valeria Maselli, Marilena Galdiero, Valeria Maselli, Marco Guida Marco Guida Antonia Feola, Emilia Galdiero, Marco Guida Marco Guida Rosa Carotenuto, Paola Cuomo, Marco Guida Rosanna Capparelli, Rosanna Capparelli, Rosa Carotenuto, Marco Guida Marco Guida Marco Guida Marco Guida Emilia Galdiero, Rosa Carotenuto, Marco Guida

Summary

Researchers found that polystyrene microplastics can worsen Candida albicans fungal infections in both cell culture and animal models. In lab tests with human intestinal cells and in wax moth larvae, the presence of microplastics promoted more severe infection outcomes. The study provides new experimental evidence that microplastic exposure may increase vulnerability to opportunistic fungal infections.

Polymers
Body Systems
Study Type In vivo

Plastic pollution is an important environmental problem, and microplastics have been shown to have harmful effects on human and animal health, affecting immune and metabolic physiological functions. Further, microplastics can interfere with commensal microorganisms and exert deleterious effects on exposure to pathogens. Here, we compared the effects of 1 µm diameter polystyrene microplastic (PSMPs) on <i>Candida albicans</i> infection in both in vitro and in vivo models by using HT29 cells and <i>Galleria mellonella</i> larvae, respectively. The results demonstrated that PSMPs could promote <i>Candida</i> infection in HT29 cells and larvae of <i>G. mellonella</i>, which show immune responses similar to vertebrates. In this study, we provide new experimental evidence for the risk to human health posed by PSMPs in conjunction with <i>Candida</i> infections.

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