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Summary
Researchers found that microplastic exposure induced kidney injury resembling Heymann nephritis in rat models and worsened gut microbial imbalance. The study identified TFF3 as a novel upstream regulator associated with increased interleukin-17A levels following microplastic exposure. These findings suggest a potential link between microplastic exposure, gut dysbiosis, and kidney inflammation in animal models.
1. Microplastic exposure induced Heymann nephritis-like glomerular injury in rat models.2. Microplastic exposure exacerbated gut dysbiosis in Heymann nephritis rat models.3. The upstream regulator TFF3 as a novel mediator was associated with the upregulation of interleukin-17A following microplastic exposure.