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Exposure to Nanoplastics Cause Caudal Vein Plexus Damage and Hematopoietic Dysfunction by Oxidative Stress Response in Zebrafish (Danio rerio)
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Researchers found that nanoplastic exposure in zebrafish embryos caused damage to blood vessel development and impaired the formation of blood cells. The nanoplastics triggered oxidative stress in vascular and blood-forming cells, disrupting normal developmental processes. The study suggests that nanoplastic contamination in aquatic environments could affect early development in fish by interfering with their circulatory and blood cell production systems.
The results of this study suggest that NPs induce oxidative stress in vascular ECs and HSPCs, which mediates CVP damage and impairs hematopoiesis in zebrafish embryos.
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Nanoplastic Exposure Mediates Neurodevelopmental Toxicity by Activating the Oxidative Stress Response in Zebrafish (Danio rerio)
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Exposure to 20-nanometer plastic particles caused developmental problems in zebrafish embryos, including shorter body length, heart defects, and lower survival rates. The nanoplastics specifically damaged the development of motor neurons and triggered oxidative stress, a harmful chemical imbalance in cells. These results suggest that very small plastic particles could interfere with early brain and nerve development.
Polystyrene Nanoplastic Exposure Induces Developmental Toxicity by Activating the Oxidative Stress Response and Base Excision Repair Pathway in Zebrafish (Danio rerio)
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Researchers exposed zebrafish embryos to polystyrene nanoplastics at various concentrations and found significant developmental abnormalities including reduced hatching rates and increased malformations. The nanoplastics activated oxidative stress responses and DNA repair pathways, indicating cellular damage during critical early development stages. The study provides mechanistic evidence for how nanoplastic exposure can disrupt normal embryonic development in aquatic organisms.
Polystyrene nanoplastics induce developmental impairments and vasotoxicity in zebrafish (Danio rerio)
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Zebrafish exposed to polystyrene nanoplastics at environmentally realistic concentrations showed developmental problems including heart defects, brain abnormalities, blood vessel damage, and reduced swimming ability. The nanoplastics accumulated on the fish's body and triggered cell damage and oxidative stress even at very low doses. Since zebrafish are commonly used to model human developmental processes, these findings raise concerns about what nanoplastic exposure could mean for early human development.
Polystyrene nanoplastics cause developmental abnormalities, oxidative damage and immune toxicity in early zebrafish development
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Zebrafish embryos exposed to polystyrene nanoplastics showed dose-dependent developmental problems including delayed hatching, reduced survival, smaller body size, and the nanoplastics accumulated in critical organs like the eyes, heart, liver, and brain. The particles triggered oxidative stress that damaged cells and activated inflammatory immune responses, demonstrating how nanoplastic contamination in water can cause widespread harm to developing organisms.
Cardiovascular toxicity assessment of polyethylene nanoplastics on developing zebrafish embryos
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Researchers assessed the cardiovascular effects of polyethylene nanoplastics on developing zebrafish embryos and found that exposure above 50 micrograms per milliliter caused pericardial edema, reduced cardiac output, and impaired blood vessel formation. The nanoplastics also triggered oxidative stress and inflammation, which contributed to blood clot formation in the embryos. The study suggests that nanoplastic exposure could pose risks to cardiovascular development in living organisms.
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