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Microplastics in the bloodstream can induce cerebral embolism and neurological impairment
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Microplastics block blood flow in the brain, mouse study reveals
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Researchers reviewed evidence for microplastic particles in human blood, examining the blood compartment as a key exposure route and biomonitoring target. The findings highlight growing concern about systemic plastic particle circulation and the need for standardized detection methods to assess human health risk.
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Researchers found that patients with carotid artery disease who had detectable microplastics and nanoplastics within their carotid plaques faced a significantly higher risk of death or major cardiovascular events compared to patients whose plaques contained no detectable plastic particles.
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This review synthesizes current evidence on how microplastics and nanoplastics (MNPs) may contribute to cardiovascular disease, examining their presence in blood, atherosclerotic plaques, thrombi, and heart tissue. Experimental studies point to oxidative stress, mitochondrial dysfunction, inflammation, and fibrotic remodeling as plausible mechanisms linking MNP exposure to heart disease, though causal relationships in humans remain unproven and standardized detection methods are still lacking.
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