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Common Chemical Plasticizer Di(2-Ethhylhexyl) Phthalate Exposure Exacerbates Coxsackievirus B3 Infection
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This study found that DEHP, a common plasticizer in PVC products, exacerbates Coxsackievirus B3 (CVB3) infection, suggesting plasticizer exposure may act as a proviral environmental factor. The findings highlight DEHP as a potential contributor to increased severity of viral infections and call for further investigation into its immune-disrupting mechanisms.
DEHP may represent a major environmental factor associated with the severity of CVB infection. Further understanding of how DEHP exacerbates infection may better elucidate its potential role as a proviral environmental factor.
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Inflammation, immune dysfunction and viral disease: could micro- and nanoplastics be the missing link?
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This review examines how tiny plastic particles in the environment might weaken immune defenses and worsen viral infections. The authors synthesize existing research showing that these particles can stabilize viruses, trigger harmful inflammation in cells, and suppress the body's antiviral responses, potentially increasing susceptibility to severe infectious diseases.
Plasticizers determine a deeper reshape of soil virome than microplastics
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Researchers compared how microplastics and the common plasticizer diethyl phthalate independently affect soil viral communities, finding that the plasticizer caused a much more dramatic shift in viral diversity than the plastic particles themselves. Diethyl phthalate exposure led to a three-fold increase in viral genetic material and triggered widespread activation of dormant viruses within soil bacteria. The findings suggest that the chemical additives leaching from plastics may pose a greater threat to soil ecosystems than the physical plastic particles.
Solving the impact of Phthalate plasticizers in relieving environment pollution
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This review examines how phthalate plasticizers—particularly DEHP, DEP, and DBP found in food packaging and cosmetics—enter soil and human bodies, where they disrupt metabolic and reproductive systems and contribute to environmental plastic pollution.
Toxic effects of polyethylene terephthalate microparticles and Di(2-ethylhexyl)phthalate on the calanoid copepod, Parvocalanus crassirostris
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Researchers tested the toxic effects of polyethylene terephthalate microparticles and the plasticizer DEHP on aquatic organisms, finding that both caused harm individually and that combined exposure produced distinct toxic responses.
Environmental factors strongly influence the leaching of di(2-ethylhexyl) phthalate from polyvinyl chloride microplastics
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Researchers found that environmental conditions like temperature, UV light, salinity, and pH strongly influence how fast the plasticizer DEHP leaches from PVC microplastics into water. Higher temperatures and UV exposure significantly accelerated the release of this endocrine-disrupting chemical. This is important because it means microplastics in warm, sunlit waters may release harmful additives much faster than lab studies under standard conditions would predict.
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When a large batch of papers lands in the Atlas, we read through it and send a short write-up of what stood out.