We can't find the internet
Attempting to reconnect
Something went wrong!
Hang in there while we get back on track
Riesgo cardiovascular femenino
AI summary Read the abstract
This review synthesizes evidence on clinical, hormonal, and environmental factors — including hormonal aging, microplastics, and environmental exposures — that influence cardiovascular disease risk in women, proposing integrated preventive strategies with a gender perspective.
Introducción: la enfermedad cardiovascular (ECV), globalmente, es la principal causa de muerte en mujeres. Los factores de riesgo incluyen envejecimiento hormonal, exposición ambiental e inequidades en el diagnóstico. Objetivo: sintetizar la evidencia disponible sobre los factores clínicos, hormonales y ambientales influyentes en el riesgo cardiovascular en mujeres, para proponer estrategias preventivas integrales y con perspectiva de género. Métodos: se realizó una revisión narrativa de la literatura en las bases de datos PubMed, Scopus y SciELO, incluyendo publicaciones entre enero de 2019 y mayo de 2024. Priorizando artículos originales, revisiones sistemáticas, guías clínicas y editoriales de consenso que abordaran población femenina. La búsqueda tuvo los términos: “cardiovascular disease in women”, “environmental pollution”, “endothelial dysfunction”, “menopause and CVD” y “microplastics cardiovascular risk”, filtros por idioma (inglés y español) y población femenina. Resultados: factores clínicos (estrés, nivel socioeconómico y características fisiopatológicas), hormonales (disminución de estrógenos y resultados adversos del embarazo) y ambientales (contaminación del aire, ruido, luz nocturna y microplásticos) interactúan para aumentar el riesgo cardiovascular en mujeres, destacando la disfunción endotelial como mecanismo central. Conclusiones: la prevención cardiovascular en mujeres requiere enfoque integral considerando la transición hormonal y entorno ambiental. La integración de políticas públicas con enfoque de género es indispensable
More Papers Like This
Risk Factors for Cardiovascular and Metabolic Disease: Integrating Traditional and Novel Paradigms
AI summary Read the abstract
This review integrates traditional cardiovascular risk factors with emerging environmental risk factors including microplastics, air pollution, and endocrine disruptors, arguing that a significant proportion of cardiovascular events occur in individuals considered low-risk by conventional models alone.
Relationship between microplastics and cardiovascular risk factors
AI summary Read the abstract
This review examines the emerging evidence linking microplastic exposure to cardiovascular risk factors, including endothelial dysfunction, oxidative stress, and inflammation, which are early indicators of heart disease. The authors conclude that microplastics represent a newly recognized environmental cardiovascular risk factor warranting further clinical research.
Microplastics and Nanoplastics in Cardiovascular Disease: An Emerging Cardiovascular Risk Factor
AI summary Read the abstract
Researchers reviewed mounting experimental and early clinical evidence that microplastics and nanoplastics cross epithelial barriers, accumulate in arterial plaques, cardiac tissue, and thrombi, and trigger oxidative stress and coagulation dysregulation, framing these particles as an emerging cardiovascular risk factor that warrants integration into disease prevention frameworks.
Micro and Nano-plastic particles: What are they and do they effect cardiovascular health?
AI summary Read the abstract
This review examines the cardiovascular health effects of micro- and nanoplastics, summarizing evidence that these particles have been detected in human tissues including arterial plaques and may promote endothelial dysfunction and inflammation. The authors call for further clinical and epidemiological research into cardiac risk.
Microplastics and Cardiovascular Disease: Should Clinicians Be Paying Attention?
AI summary Read the abstract
This clinical review summarizes evidence for microplastics as a cardiovascular risk factor, noting that they have been detected in human cardiovascular tissues and that in vitro and animal studies link them to oxidative stress, endothelial dysfunction, and platelet disruption, while cautioning that human evidence remains associative.
Research digests by email
When a large batch of papers lands in the Atlas, we read through it and send a short write-up of what stood out.