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Article ? AI-assigned paper type based on the abstract. Classification may not be perfect — flag errors using the feedback button. Tier 2 ? Original research — experimental, observational, or case-control study. Direct primary evidence. Environmental Sources Human Health Effects Reproductive & Development Sign in to save

Placental-fetal distribution of carbon particles in a pregnant rabbit model after repeated exposure to diluted diesel engine exhaust

Particle and Fibre Toxicology 2023 13 citations ? Citation count from OpenAlex, updated daily. May differ slightly from the publisher's own count. Score: 50 ? 0–100 AI score estimating relevance to the microplastics field. Papers below 30 are filtered from public browse.
Eva Bongaerts, Tim S. Nawrot, Flemming R. Cassee Pascale Chavatte‐Palmer, Eva Bongaerts, Congrong Wang, Congrong Wang, Marcel Ameloot, Hannelore Bové, Maarten B. J. Roeffaers, Maarten B. J. Roeffaers, Pascale Chavatte‐Palmer, Tim S. Nawrot, Anne Couturier‐Tarrade, Marcel Ameloot, Maarten B. J. Roeffaers, Flemming R. Cassee Tim S. Nawrot, Flemming R. Cassee Tim S. Nawrot, Flemming R. Cassee Tim S. Nawrot, Tim S. Nawrot, Flemming R. Cassee

Summary

Researchers confirmed that carbon particles from diesel exhaust inhaled by pregnant rabbits travel from the lungs through the placenta and into fetal organs. This demonstrates that airborne particles — including nanoplastics and combustion soot — can cross the placental barrier during pregnancy, potentially altering fetal development.

The results confirmed the translocation of maternally inhaled CPs from diesel engine exhaust to the placenta which could be detected in fetal organs during late-stage pregnancy. The exposed can be clearly discriminated from the control group with respect to fetoplacental biometry and CP load. The differential particle load in the fetal organs may contribute to the effects on fetoplacental biometry and to the malprogramming of the fetal phenotype with long-term effects later in life.

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