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Knockdown of LCN2 Attenuates Brain Injury After Intracerebral Hemorrhage via Suppressing Pyroptosis

Neuropsychiatric Disease and Treatment 2024 5 citations ? Citation count from OpenAlex, updated daily. May differ slightly from the publisher's own count. Score: 55 ? 0–100 AI score estimating relevance to the microplastics field. Papers below 30 are filtered from public browse.
Yangyang Zhao, Qiuxiang Xiao, Tao Sun, Haiyun Yu, Muyun Luo

Summary

Researchers found that reducing levels of a protein called lipocalin-2 (LCN2) in the brain lessened injury severity after intracerebral hemorrhage in animal models. The protective effect was linked to suppression of pyroptosis, a form of inflammatory cell death. The study suggests that targeting LCN2 could be a potential strategy for reducing brain damage following hemorrhagic stroke.

Body Systems

Inhibition of LCN2 attenuates brain injury after ICH via suppressing pyroptosis, which provide guidance for ICH management.

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