We can't find the internet
Attempting to reconnect
Something went wrong!
Hang in there while we get back on track
P21-20 Lung accumulation of polyethylene microplastics may cause chronic inflammatory response
No summary available — this paper's abstract is not included in the open metadata provided by the publisher. Learn why →
More Papers Like This
Microplastic Accumulation in the Human Lung: Distinct Signatures in Lung Cancer vs Normal Tissue
AI summary Read the abstract
Scientists found tiny plastic particles (from things like packaging and bottles) in all the lung tissue samples they studied, both cancerous and healthy. Cancer samples tended to have more plastic bits, especially smaller fiber-shaped pieces made of common plastics like polypropylene and PET, but this was a small study, so it can't yet prove plastics cause cancer, just that they're worth investigating further. Bottom line: this adds to growing evidence that microplastics are showing up in our bodies, and researchers want to know if they're contributing to lung disease.
Toxicological effects of different particle sizes and types of nano-plastics (NPs) on human lung cells: implications for the health risk of airborne microplastic pollution
AI summary Read the abstract
Researchers exposed human A549 alveolar cells to polystyrene and polymethylmethacrylate nanoplastics of 30 and 50 nm and found that smaller size and higher concentration produced stronger cytotoxicity, oxidative stress, inflammation, and apoptosis—with PS nanoparticles more toxic than PMMA—providing in vitro evidence of respiratory health risks from inhaled airborne nanoplastics.
Causal relationship between micro- and nanoplastics and lung function
AI summary Read the abstract
Researchers quantified micro- and nanoplastic levels in whole blood from young adults using pyrolysis GC-MS and measured lung function by spirometry, analyzing the causal relationship between circulating plastic particle burden and forced expiratory volume and vital capacity outcomes.
Respiratory risks of microplastics and nanoplastics: Where? What? How?
AI summary Read the abstract
Researchers systematically reviewed experimental and epidemiological evidence linking inhaled microplastics and nanoplastics to pulmonary disorders including COPD, asthma, and fibrosis, identifying oxidative stress as the dominant initiating mechanism that cascades into mitochondrial dysfunction, inflammation, barrier disruption, and multiple forms of cell death.
Research digests by email
When a large batch of papers lands in the Atlas, we read through it and send a short write-up of what stood out.