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Investigating the hypothesis role of the hidden poison microplastics in lymphoma development
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This review explores the hypothesis that microplastics, which are found throughout the environment and enter the human body through ingestion, inhalation, and skin contact, could indirectly contribute to the development of lymphoma. Researchers examined how microplastics cause oxidative stress, immune disruption, and metabolic changes that may create conditions favorable to this type of blood cancer. While direct evidence is still lacking, the study suggests that the mechanisms warrant further investigation.
Abstract Microplastics, ranging from 1 micron to 5 mm in size, ubiquitous environmental pollutants, arise from diverse sources such as industrial processes, personal care products, textiles, and urban runoff, permeating air, soil, freshwater, sediment, and marine environments. Human exposure occurs through ingestion, inhalation, and dermal contact, leading to health concerns including oxidative stress, metabolic disruption, and immune modulation, with potential direct links to lung, skin, and digestive cancers. While direct evidence linking microplastics to lymphoma remains elusive, therefore we hypothesized that bloodstream's immediate exposure post-entry suggests a plausible indirect link. This review explores mechanisms around our hypothesis by which microplastics might contribute to lymphoma, encompassing inflammation, oxidative stress, immune dysregulation, endocrine disruption, and genotoxicity. Understanding these pathways is crucial given lymphoma's global impact. Future research warrants comprehensive epidemiological studies and interdisciplinary approaches, integrating epidemiology, experimental research, and computational modeling to elucidate the association between microplastics and lymphoma.
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Microscopic menace: exploring the link between microplastics and cancer pathogenesis
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This review examines the growing evidence linking microplastic exposure to cancer development in humans. Microplastics can accumulate in the body and trigger inflammation, oxidative stress, and other biological changes associated with tumor growth. While more clinical research is needed, the review highlights that microplastics should be taken seriously as a potential factor in cancer risk.
The hidden poison - microplastic : Inflammatory catalyst of cancer development
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The hidden threat: Microplastics and cancer biology
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This review synthesizes current research on the potential links between micro- and nanoplastic exposure and cancer biology, examining how these particles may contribute to tumor development in organs including the gut, lungs, skin, liver, kidney, and brain. The study suggests that chronic exposure to plastic particles can trigger molecular changes such as oxidative stress, inflammation, and DNA damage that are associated with cancer initiation, though clear causal evidence in humans is still lacking.
From Exposure to Oncogenesis: the Role of Microplastics and Associated Pollutants in Cancer - a Literature Review
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This literature review examined the growing evidence linking microplastic exposure to cancer development. Microplastics have been found in human lung, liver, and colon tissue, and research suggests they may promote cancer through chronic inflammation, oxidative stress, and by carrying known carcinogens like heavy metals and persistent organic pollutants into the body.
Micro(nano)plastics pollution and human health: How plastics can induce carcinogenesis to humans?
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This review examines how microplastics and nanoplastics enter the human body through food, water, and air, and how they may contribute to cancer development. Common plastic types like polystyrene and PVC, along with toxic chemicals they carry such as PAHs and PCBs, have been linked to DNA damage, oxidative stress, and inflammation, all of which can promote cancer. The paper highlights that while the evidence is growing, more research is needed to understand the full cancer risk from chronic microplastic exposure.
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When a large batch of papers lands in the Atlas, we read through it and send a short write-up of what stood out.